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CALM1 — GPI

Text-mined interactions from Literome

Lustig et al., Biochim Biophys Acta 1992 : The increase in [ Ca2+ ] i elicited by ATP apparently caused the activation of a calmodulin dependent phospholipase A2 since trifluoperazine, an inhibitor of calmodulin , and quinacrine, an inhibitor of phospholipase A2, prevented the stimulation of PGI2 release by ATP
Satoh et al., Biochem Biophys Res Commun 1985 : The results suggest that angiotensin II may enhance the intracellular Ca2+ level through the influx of extracellular Ca2+ and then, calmodulin activated with Ca2+ will stimulate both PGE2 and PGI2 productions via its activation of phospholipase A2 in the dog renal arteries