Cushing et al., FASEB J 2008
(Fibrosis...) :
Using an exogenous Smad-responsive transcriptional promoter reporter, we show that Smad activity is repressed by FGF-2 , likely an effect of the fact that FGF-2 treatment prevents the nuclear localization of Smads in these cells
Miyoshi et al., Mol Cell Endocrinol 2010
:
Interestingly, FGF-8 enhanced BMP induced Smad1/5/8 and Id-1-promoter activities with decreased expression of Smad6/7
Stuhlmiller et al., Development 2012
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Our results identify an early requirement for FGF/MAPK signaling in amniote neural crest induction and suggest an intriguing role for FGF mediated Smad inhibition in ectodermal development