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FGF2 — IL8

Text-mined interactions from Literome

Bodo et al., Eur J Cell Biol 1999 (Craniofacial Dysostosis) : ELISA showed that bFGF induced interleukin secretion differed in normal and Crouzon osteoblasts
Giri et al., Am J Pathol 2001 (Prostatic Hyperplasia) : Interleukin-8 is a paracrine inducer of fibroblast growth factor 2 , a stromal and epithelial growth factor in benign prostatic hyperplasia ... Therefore IL-8 produced by prostatic epithelial cells can induce FGF2 , a potent stromal and epithelial growth factor, and in this manner promote the abnormal proliferation of the prostatic transition zone that is critical in the pathogenesis of BPH
Castro et al., Prostate 2004 (Prostatic Hyperplasia) : IL-8 is also present at increased levels in BPH tissues and induces expression of FGF2 , a potent stromal growth factor
Im et al., J Biol Chem 2007 : bFGF stimulation of MMP-13 was mediated at the transcriptional level and, at least in part, by stimulation of interleukin-1 production
Ulbrich et al., J Cell Biochem 2008 : After 24 h micro g, bFGF elevated fibronectin, Flk-1 and Flt-1 protein, and reduced IL-6 and IL-8 compared with vehicle treated micro g cultures
Sandell et al., Osteoarthritis Cartilage 2008 (Arthritis) : In the presence of IL-1beta, FGF-18 increased expression of ADAMTS-4, aggrecan, BMP-2, COL2A1, CCL3, CCL4, CCL20, CXCL1, CXCL3, CXCL6, IL-1beta, IL-6, and IL-8 and decreased ADAMTS-5, MMP-13, CCL2, and CCL8
Herseth et al., Particle and fibre toxicology 2008 : Silica exposure induced an increase of IL-8 release from monocytes and from pneumocytes alone, and the FGF-2 level in the medium increased upon silica exposure of pneumocytes
Nakayama et al., J Oral Sci 2012 : In human osteoblast-like Saos2 cells, FGF2 increased the expression of the osteopontin ( SPP1, 16.7-fold ), interleukin-8 ( IL8, 6.4-fold ) and IL11 ( 4.8-fold ) genes
Chandrasekhar et al., J Cell Physiol 1989 : Induction of interleukin-1 receptors on chondrocytes by fibroblast growth factor : a possible mechanism for modulation of interleukin-1 activity
Iwamoto et al., Immunol Lett 1989 : Furthermore, interleukin 1 (IL-1) suppressed by 50-60 % the incorporation of [ 3H ] thymidine into DNA in high density chondrocyte cultures on plastic dishes in the presence of 10 % serum or fibroblast growth factor , although it increased twofold this incorporation in 0.3 % serum alone
Norioka et al., Jpn J Cancer Res 1994 (Neovascularization, Pathologic) : Interaction of interleukin-1 and interferon-gamma on fibroblast growth factor induced angiogenesis
Tamura et al., Endocrinology 1996 (Arthritis...) : Nitric oxide mediates interleukin-1 induced matrix degradation and basic fibroblast growth factor release in cultured rabbit articular chondrocytes : a possible mechanism of pathological neovascularization in arthritis
Nakamura et al., J Hypertens 1996 : bFGF , TGF-beta, interleukin-1 alpha, -1 beta and -6 stimulated DNA synthesis in VSMC significantly, whereas HGF did not
Schwarz et al., Kidney Int 1997 : Finally, unlike other mesangial growth factors basic fibroblast growth factor (bFGF) induced MCP-1, RANTES, and IL-8 mRNA expression, suggesting an involvement of autocrine regulation mechanisms in mesangial chemokine expression
Kumar et al., Int J Oncol 1998 : The organization of endothelial cells to form tubes on Matrigel was induced by bFGF and, to a lesser extent, by VEGF/VPF and IL-8
Presta et al., J Biol Chem 1998 : IL-8 did not prevent the binding of bFGF to its tyrosine kinase FGF receptors ( FGFRs ) nor to cell surface heparan sulfate proteoglycans ( HSPGs ) ... A transient interaction of IL-8 with the cell before the addition of the growth factor was sufficient to prevent bFGF activity
Sasaki et al., J Biochem 1998 : Nitric oxide mediates interleukin-1 induced gene expression of matrix metalloproteinases and basic fibroblast growth factor in cultured rabbit articular chondrocytes