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CXCL1 — GRAP2
Text-mined interactions from Literome
Wang et al., J Biol Chem 2001
(Cell Transformation, Neoplastic) :
We show here that either exogenous addition or continuous expression of
MGSA/GROalpha in immortalized melanocytes
enhances NF-kappaB activation, as well as mitogen activated protein ( MAP ) kinase kinase kinase ( MEKK) 1, MAP kinase kinase (MEK) 3/6, and
p38 MAP kinase activation
Petrai et al., Int J Biochem Cell Biol 2008
:
In human microvascular endothelial cells, which selectively express CXCR3-B, in a cell cycle dependent fashion,
CXCL10 and CXCL4
increased the enzymatic activity of
p38 ( MAPK )
Chen et al., Molecular vision 2011
:
Inhibition of
p38MAPK , phosphoinositide 3-kinase (PI3K)-Akt and nuclear factor-kappaB ( NF-?B ), with the inhibitors SB203580, LY294002 and pyrrolydine dithiocarbamate ( PDTC ) respectively,
reduced IL-17 ( 100 ng/ml ) mediated production of
CXCL8 , CCL2, and IL-6 in a concentration dependent manner
Dumitru et al., J Leukoc Biol 2012
(Head and Neck Neoplasms...) :
Most importantly, HNC induced
p38-MAPK activation strongly
stimulates the release of CCL4,
CXCL8 , and MMP9 by neutrophils
Blume et al., Eur Respir J 2013
(Asthma...) :
Inhibition of the extracellular signal regulated kinase 1/2 and
p38 mitogen activated protein kinase pathways selectively
blocked apical
CXCL8/IL-8 release via a post-transcriptional mechanism