◀ Back to PDCD11
IL2 — PDCD11
Text-mined interactions from Literome
Di Renzo et al., Arch Dermatol Res 2003
(Chronic Disease...) :
This enhanced
programmed cell death could not be
prevented by
IL-2
Matsui et al., Intensive Care Med 2012
:
The production of tumor necrosis factor-alpha ( TNF-a ),
interleukin-10 (IL-10), and NO and the
activation of
NF-?B p65 were reduced by hypothermia, but augmented by hyperthermia at 3-6, 24-48, 48, and 0.5 h, post-treatment initiation, respectively
Byun et al., Biochem Biophys Res Commun 2012
(Inflammation) :
In addition, EGCG treated DCs inhibited lipopolysaccharide (LPS) induced production of pro-inflammatory cytokines ( tumor necrosis factor [TNF ] -a,
interleukin [ IL]-1ß, and IL-6 ) and
activation of mitogen activated protein kinases ( MAPKs ), e.g., extracellular signal regulated kinase 1/2 ( ERK1/2 ), p38, c-Jun N-terminal kinase (JNK), and
nuclear factor ?B ( NF-?B ) p65 translocation through 67LR
Wilson et al., Nature 1994
:
Interleukin-1 beta converting enzyme ( ICE ) processes an inactive precursor to the proinflammatory cytokine,
interleukin-1 beta, and may
regulate programmed cell death in neuronal cells
Huang et al., Int J Hematol 1993
(Leukemia, Lymphocytic, Chronic, B-Cell...) :
Interleukin-2 prevents
programmed cell death in chronic lymphocytic leukemia cells
Deng et al., Proc Natl Acad Sci U S A 1993
:
Absence of
interleukin 2 (IL-2) from IL-2 dependent cells, such as the cytotoxic T-cell line CTLL2,
causes DNA fragmentation and
programmed cell death ( apoptosis )
Tsuda et al., Jpn J Cancer Res 1993
(Leukemia, T-Cell) :
Interleukin-2 prevents
programmed cell death in adult T-cell leukemia cells