◀ Back to PI3

PI3 — RET

Text-mined interactions from Literome

Melillo et al., Oncogene 2001 : Tyrosine 1062 of Ret, which represents an intracytoplasmic docking site for multiple signaling molecules, is essential for Ret mediated activation of phosphatidylinositol 3-Kinase (PI3-K)
Tang et al., Dev Biol 2002 : Activation of RET resulted in increased levels of phosphatidylinositol 3-kinase (PI3K) activity and Akt/PKB phosphorylation
Pelicci et al., Mol Cell Biol 2002 (Multiple Endocrine Neoplasia Type 2a) : Overexpressed Rai resulted in the potentiation of the Ret dependent activation of phosphatidylinositol 3-kinase (PI3K) and Akt
Miyagi et al., Mol Carcinog 2004 (Cell Transformation, Neoplastic...) : Chronic expression of RET/PTC 3 enhances basal and insulin stimulated PI3 kinase/AKT signaling and increases IRS-2 expression in FRTL-5 thyroid cells