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CD2 — FGFR3

Text-mined interactions from Literome

Toth et al., Neurotoxicology 1987 : 100 microM Pb+2 and Cd+2 also induced a release of ACh from the permeabilized synaptosomes
Colasante et al., J Neurocytol 1995 : ACh release was stimulated in cutaneous pectoris nerve-muscle preparations by : ( 1 ) 1 mM Cd2+ in Ca ( 2+ ) -free medium for a period of 3 h, ( 2 ) 25 or 40 mM K+ in normal Ringer 's solution
Tamura et al., Brain Res 1995 (Ion Channel Gating) : Cd2+ markedly blocked the release of both DA and ACh , Co2+ and Ni2+ slightly blocked the release of DA, and the release of ACh was not blocked by these two divalent cations ... These results suggest that the high K ( + ) -evoked release of retinal DA is largely mediated by omega-agatoxin IVA and omega-conotoxin MVIIC sensitive calcium channels ( probably Q-type channels ), while the release of retinal ACh is largely mediated by as yet uncharacterized Cd2+ sensitive calcium channels
Colasante et al., Neuromuscul Disord 1993 : By contrast, an intense immunofluorescence of the NMJ was observed after an exhaustive ACh release induced by Cd2+ in Ca ( 2+ ) -free medium, which leads to a synaptic vesicle depletion and an increase in the membranous structures in nerve terminals