◀ Back to MYD88
IL23A — MYD88
Text-mined interactions from Literome
Marta et al., Eur J Immunol 2008
(Encephalomyelitis, Autoimmune, Experimental) :
Our data suggest that
MyD88 mediates the induction of mDC IL-6 and
IL-23 responses after MOG immunization, which in turn drives IL-17 producing encephalitogenic Th17 cell activation
Ma et al., Infect Immun 2010
(Pneumococcal Infections) :
Furthermore, morphine treatment impaired
MyD88 dependent
IL-23 production in alveolar macrophages and dendritic cells in response to in vitro S. pneumoniae cell infection
Keestra et al., Infect Immun 2011
(Colitis...) :
Expression of
IL-23 in the cecal mucosa during S. Typhimurium colitis was
dependent on the presence of
MyD88
Ather et al., J Immunol 2011
(Disease Models, Animal...) :
Furthermore, SAA drives production of IL-1a, IL-1ß, IL-6,
IL-23 , and PGE ( 2 ), causes dendritic cell ( DC ) maturation, and
requires TLR2,
MyD88 , and the NLRP3 inflammasome for secretion of IL-1ß by DCs and macrophages
Dejima et al., Infect Immun 2011
(Candidiasis) :
Early
IL-23 production in a
TLR2/MyD88 dependent manner and IL-23 triggered tyrosine kinase 2 (Tyk2) signaling were essential for IL-17A production by ?d T cells