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ICAM1 — IL37
Text-mined interactions from Literome
Giavazzi et al., Cancer Res 1992
(Melanoma) :
Tumor necrosis factor,
interleukin 1, and gamma-interferon also
caused the release of soluble
ICAM-1
Andersen et al., Nephrol Dial Transplant 1992
:
Furthermore, the in vitro model indicates that
ICAM-1 expression is
regulated by gamma-interferon and
interleukin-1 produced by activated T lymphocytes and macrophages
Stuyt et al., Immunology 2003
:
Selective
regulation of
intercellular adhesion molecule-1 expression by interleukin-18 and
interleukin-12 on human monocytes
Moreira et al., Am J Pathol 2006
(Disease Susceptibility...) :
Expression of
ICAM-1 was up-regulated on T lymphocytes after infection with the fungus, and its expression was
dependent on interferon-gamma, tumor necrosis factor-alpha, and
interleukin-12
Krutmann et al., J Invest Dermatol 1990
:
Stimulation of cells with recombinant human ( rh
) interleukin (IL) 1 alpha, rhIL-4, rhIL-5, rhIL-6, rh granulocyte/macrophage colony stimulating factor ( GM-CSF ), rh interferon alpha ( rhIFN alpha ), and rh transforming growth factor beta ( TGF beta ) did not
increase ICAM-1 surface expression
Giwa et al., Neurology 2012
(Cerebral Small Vessel Diseases) :
Endothelial dysfunction is a possible causal factor, and circulating markers of endothelial
activation (
intercellular adhesion molecule-1 , thrombomodulin ) and inflammation (
interleukin [ IL]-6 ) are elevated in patients with SVD
Sano et al., Mol Cell Biochem 1994
:
Cellular expression of
ICAM-1 was drastically
induced by TNF or
interleukin-1 stimulation, and the moderate expression with delayed-action was observed only by lipopolysaccharide stimulation
Wake et al., Blood 1995
(Bone Resorption...) :
We report the following novel features of homotypic adhesion via leukocyte function associated antigen-1 ( LFA-1 ) /intracellular adhesion molecule-1 ( ICAM-1 ) pathway that suggest a role for it in cytokine production and rapid proliferation of ATL cells : ( 1 ) ATL cells show clustering in a calcium dependent manner, even at the higher concentration ; ( 2 ) ATL cells consistently and highly express ICAM-1 and an active form of LFA-1, whereas integrin expression, except for LFA-1, is rather lower compared with that of normal CD4+ T cells ; ( 3 ) ATL cells make conjugate formation within 6 minutes and clustering within 48 hours, both of which are inhibited by the addition of monoclonal antibodies ( MoAbs ) against LFA-1 and ICAM-1 ; ( 4 ) spontaneous mRNA transcription and protein secretion of both
interleukin-1 and parathyroid hormone related protein are observed consistently in ATL cells, and these productions are
inhibited by anti-LFA-1 and
anti-ICAM-1 MoAbs but are markedly increased by cross linking of LFA-1 and ICAM-1 by the immobilized specific MoAbs ; and ( 5 ) proliferative responses of ATL cells are also inhibited by these MoAbs
Campbell et al., J Urol 1994
(Carcinoma, Transitional Cell...) :
In contrast,
interleukin-1 and phorbol myristate acetate exhibited variable
effects on
ICAM-1 expression, and interferon-alpha had no effect
Shrikant et al., J Neuroimmunol 1994
(Encephalomyelitis, Autoimmune, Experimental) :
Regulation of
intercellular adhesion molecule-1 gene expression by tumor necrosis factor-alpha,
interleukin-1 beta, and interferon-gamma in astrocytes
Deisher et al., Life Sci 1993
(Second Messenger Systems) :
Surface protein expression of vascular cell adhesion molecule-1, endothelial leukocyte adhesion molecule-1, or
intercellular adhesion molecule-1 , which is
induced by tumor necrosis factor,
interleukin-1 , and lipopolysaccharide, was not induced by pentoxyfilline, a phosphodiesterase inhibitor, nor by dibutyryl cyclic adenosine monophosphate
Lai et al., Int J Cancer 1993
(Fibrocystic Breast Disease) :
Possible
regulation of soluble
ICAM-1 levels by
interleukin-1 in a sub-set of breast cysts
Baumgartner-Parzer et al., Diabetologia 1995
:
Stimulation of confluent HUVECs, kept in 30 vs 5 mmol/l glucose for 13 +/- 1 days, with 20 U/ml
interleukin-1 for 24 h ( ICAM-1 ) and 4 h ( endothelial leukocyte adhesion molecule 1 )
resulted in reduced
ICAM-1 ( 84.8 +/- 27.0 %, p < 0.05 ) and endothelial leukocyte adhesion molecule-1 ( 87.6 +/- 22.4 %, p < 0.05 ) expression vs control cells, while that of PECAM ( t : 24 h ) and vascular cell adhesion molecule-1 ( t : 16 h ) remained unchanged
Perretti et al., Biochem Biophys Res Commun 1996
:
This opposite modulatory
role of
interleukin-1 and dexamethasone on
intercellular adhesion molecule-1 expression was also, for the first time, observed in vivo using mouse peritoneal macrophages : a four-fold increase in intercellular adhesion molecule-1 expression was measured after local administration of the cytokine ( 5 micrograms/kg ) and this effect was greatly inhibited ( > 70 % ) by co-injection with 1 microgram dexamethasone
Ikeda et al., J Am Soc Nephrol 1996
:
In an enzyme linked immunosorbent assay,
interleukin 1 beta ( IL-1 beta ; 10 ng/mL )
increased ICAM-1 molecule expression on cultured rat mesangial cell surface in a time dependent manner
Wang et al., Circ Res 1998
:
Cardiac graft intercellular adhesion molecule-1 ( ICAM-1 ) and
interleukin-1 expression mediate primary isograft failure and
induction of
ICAM-1 in organs remote from the site of transplantation