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ATP5O — AVP

Text-mined interactions from Literome

Laroche-Joubert et al., American journal of physiology. Renal physiology 2000 (Potassium Deficiency) : In CCDs, K ( + ) -ATPase was stimulated by calcitonin and isoproterenol in NK rats ( type I K ( + ) -ATPase ) and by dD-AVP in LK rats ( type III K ( + ) -ATPase ) ... In OMCDs, dD-AVP and glucagon stimulated type III but not type I K ( + ) -ATPase
Mordasini et al., American journal of physiology. Renal physiology 2005 : Time course experiments revealed that in response to AVP , the cell surface expression of both endogenous Na-K-ATPase and hybrid Na pumps containing a c-myc tagged wild-type human alpha ( 1 ) -subunit increased transiently ... Therefore, stimulation of Na-K-ATPase cell surface expression by AVP is dependent on PKA but does not rely on alpha ( 1 ) -subunit phosphorylation on serine 943 in the collecting duct principal cells
Sakuma et al., Hypertens Res 2005 (Dehydration) : Since arginine vasopressin (AVP) regulates Na-K-ATPase activity directly via protein kinase A and indirectly via hyperosmolality, we investigated the acute and chronic effects of hyperosmolality on Na-K-ATPase and AVP dependent cAMP generation in the MAL and OMCD
Charlton et al., J Endocrinol 1990 : Stimulation of rat renal medullary Na+/K ( + ) -ATPase by arginine vasopressin is mediated by the V2 receptor ... In contrast, 100 pmol of the V2/V1 antagonist/l significantly ( P less than 0.001 ) inhibited the stimulation of Na+/K ( + ) -ATPase activity by 1 fmol AVP/l from 55.5 +/- 4.3 ( S.E.M. ) to 31.9 +/- 1.6 mean integrated extinction ( MIE ) after 4 min of stimulation and from 67.0 +/- 3.2 to 36.9 +/- 0.7 MIE after 10 min of stimulation ... We conclude that the stimulation of Na+/K ( + ) -ATPase by AVP is mediated by the V2 receptor in the rat renal MTAL
Charlton et al., J Endocrinol 1990 : Previous studies have indicated that ornithine decarboxylase (ODC) may be involved in the stimulation of Na+/K ( + ) -ATPase activity by arginine vasopressin (AVP) in the rat renal medullary thick ascending limb of Henle 's loop ... The specific inhibitors of spermidine and spermine synthase, bis-cyclohexylammonium sulphate and N-alkylated-1,3-diaminopropane respectively, inhibited the stimulation of Na+/K ( + ) -ATPase by AVP , this inhibition being reversed by spermine
Charlton et al., J Endocrinol 1989 : Inhibition of arginine vasopressin stimulated Na+ K+ ATPase activity by difluoromethyl ornithine in the rat renal medulla ... The effect of difluoromethyl ornithine ( DFMO ), a specific inhibitor of ODC activity, on AVP stimulated Na+ K+ ATPase activity was evaluated using a cytochemical bioassay ... The addition of 20 mmol DFMO/l to the culture medium containing AVP in the concentration range 0.001-10 fmol/l inhibited completely the stimulation of Na+ K+ ATPase activity by AVP ... The response of Na+ K+ ATPase to increasing doses of ATP ( 10-40 g polypeptide/l ) was not influenced by the addition of 20 mmol DFMO/l to the culture medium containing AVP, suggesting that the prevention of AVP stimulated Na+ K+ ATPase activity by DFMO was not due to a direct effect on the enzyme
Pippard et al., Cell Biochem Funct 1986 : Complete inhibition of AVP stimulated Na+-K+-ATPase activity occurred at a hypothalamic extract concentration of 10 ( -3 ) U ml-1
Baylis et al., J Endocrinol 1986 : Infusion of hypertonic saline into six normal volunteers caused an increase in plasma osmolality from 286.8 +/- 1.7 ( mean +/- S.E.M. ) to 307.6 +/- 2.6 mosmol/kg ( P less than 0.001 ), a 7.1 % increase in estimated blood volume, a rise in plasma immunoreactive arginine vasopressin (AVP) concentrations from 1.3 +/- 0.2 to 12.7 +/- 3.6 pmol/l ( P less than 0.001 ) but no change in plasma AVP concentrations ( 2.1 +/- 0.9 and 1.9 +/- 1.3 pmol/l ) as measured by a cytochemical technique based on the ability of AVP to stimulate rat renal medullary Na+/K+-ATPase activity ... The results suggest that infusion of hypertonic saline interfered with the cytochemical assay for AVP by inhibiting AVP stimulated medullary Na+/K+-ATPase activity
Pippard et al., Clin Sci (Lond) 1984 : Synthetic [arginine]vasopressin [ ( Arg ] VP ) stimulated ATPase activity in the thick tubular cells of the renal outer medulla over the concentration range 0.01-10 fmol/l, but failed to affect ATPase activity in the proximal and distal convoluted tubules of the cortex
Smith et al., Biosci Rep 1984 (Dehydration) : The total Na+/K+ ATP-ase activity of the thick ascending limb of the loop of Henle may be stimulated by arginine-vasopressin (AVP)
Coutry et al., J Membr Biol 1995 : These results demonstrate that, in the CCD, aldosterone and AVP act synergistically to increase not only the apical sodium entry but also the basolateral Na ( + ) -K ( + ) -ATPase transport capacity : AVP allows a rapid recruitment and/or activation of an aldosterone dependent pool of latent Na ( + ) -K ( + ) -ATPase
Blot-Chabaud et al., J Membr Biol 1996 : The aim of this study was to investigate whether this AVP induced increase in basolateral Na+-K+-ATPase could depend on a dephosphorylation process ... These data suggest that PP2A is implied in the regulation of Na+-K+-ATPase activity by AVP in the CCD and that the AVP dependent increase in the number of Na+-K+-ATPases is mediated by a PP2A dependent dephosphorylation process
Barreto-Chaves et al., Kidney Int 1997 : Luminal arginine vasopressin stimulates Na ( + ) -H+ exchange and H ( + ) -ATPase in cortical distal tubule via V1 receptor