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CD58 — TNF

Text-mined interactions from Literome

Swerlick et al., J Invest Dermatol 1991 : ICAM-1, but not LFA-3 or CD44, was increased in expression in a dose- and time dependent manner by interleukin 1 (IL-1) alpha, tumor necrosis factor (TNF) alpha, lipopolysaccharide (LPS), or interferon (IFN) gamma
Ebert et al., Am J Physiol Gastrointest Liver Physiol 2009 (MAP Kinase Signaling System) : Although both TNF-alpha and IFN-gamma increased IL-8 synthesis and CD58 expression by the HT-29 cells, only IFN-gamma reduced IL-8 production by IELs
Kvale et al., J Hepatol 1993 : ICAM-1, but not LFA-3 , was rapidly up-regulated by interleukin-1 (IL-1), tumor necrosis factor-alpha (TNF) , and to some extent by interferon-gamma (IFN) and IL-6, whereas IL-4 had variable low effects, if any
Lutter et al., Adv Exp Med Biol 1995 : In preliminary experiments with fluorescence microscopy we found that neither TNF-alpha nor IL-1 beta induce LFA3 in the same fashion as IFN-gamma
Iwata et al., Curr Eye Res 1997 : Neither IFN-gamma nor TNF-alpha had any effect on LFA-3 expression on the cultured HCE cells
Labuda et al., Immunology 1998 : This response profile differs from that observed in B7 or LFA-3 costimulated T cells because our previous results showed that B7-CD28 costimulation was accompanied by high levels of IL-2, IFN-gamma and TNF, whereas LFA-3 was a potent inducer of IFN-gamma and TNF , but had little influence on IL-2 production