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CSF2 — PTX3

Text-mined interactions from Literome

Poulakis et al., Respir Med 1999 (Lung Diseases) : The aim of this study was to investigate the effects of pentoxifylline ( PTX ) on the production of TNF-alpha, IL-1 beta, IL-6 and GM-CSF by lipopolysaccharide (LPS) stimulated alveolar macrophages ( AM ) ... In PBM cultures, PTX significantly suppressed the production of TNF-alpha and GM-CSF , at all tested concentrations
Nauta et al., Kidney Int 2005 : In addition, activation of PTECs with IL-17 and CD40L, respectively, but not with IL-6 or IL-4, resulted in strongly increased production of PTX3, whereas granulocyte macrophage-colony stimulating factor ( GM-CSF ) inhibited IL-1 induced PTX3 production
Tsujino et al., J Neuroimmunol 1997 (Paraparesis, Tropical Spastic) : To clarify if pentoxifylline ( PTX ) may have therapeutic potential for human T-cell lymphotropic virus type I ( HTLV-I ) -associated myelopathy ( HAM ), we investigated the in vitro effect of PTX on spontaneous proliferation of peripheral blood lymphocytes ( SPP ), as well as on the expression of adhesion molecules, such as lymphocyte function antigen-1 ( LFA-1 ) and very late activation antigen-4 (VLA-4), and the production of inflammatory cytokines, such as tumor necrosis factor-alpha (TNF-alpha), interferon-gamma (IFN-gamma) and granulocyte-monocyte colony stimulating factor ( GM-CSF ), in cultured PBMC from 10 HAM patients, compared with control subjects
Schratzberger et al., Immunopharmacology 1999 : In the present study, we investigated the effects of the anti-inflammatory drug pentoxifylline ( PTX ) on activation of endothelial cells for enhanced adhesion and transmigration of neutrophils by lipopolysaccharide (LPS), tumor necrosis factor-alpha (TNF), interleukin-1 (IL-1) and granulocyte colony stimulating factor ( G-CSF )