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CTNNB1 — FGF7

Text-mined interactions from Literome

Treichel et al., Genes Dev 2003 : We present evidence that mBtd is required during early limb development to maintain, but not to initiate Wnt/beta-catenin dependent FGF , Shh, and BMP mediated signaling
Maegawa et al., Development 2006 : By contrast, when FGF signaling is inhibited, beta-catenin did not induce goosecoid and chordin, repress bmp4 expression or induce a dorsal axis ... These results demonstrate that FGF signaling is necessary for beta-catenin induction of the zebrafish organizer
Katoh et al., Cancer Biol Ther 2007 (Helicobacter Infections...) : WNT-beta-catenin-TCF/LEF signaling induces upregulation of MYC, CCND1, WISP1, FGF20 , JAG1 and DKK1 genes
Ambrosetti et al., Mol Cell Biol 2008 : FGF did not significantly affect the activation of beta-catenin , although it reduced both the expression of TCF/LEF factors and their induction by Wnt. Microarray analysis using osteoblasts treated with Wnt3a and FGF alone or in combination showed that about 70 % of the genes induced by Wnt3a were downregulated by combined FGF treatment
Kao et al., Eye Contact Lens 2010 (Corneal Diseases...) : On doxycycline induction, overexpression of FGF7 by corneal epithelial cells of bitransgenic Krt12-rtTA/tet-o-FGF7 mice caused nuclear translocation of beta-catenin and epithelium hyperplasia resembling human ocular surface squamous neoplasia ; in triple transgenic mice ( Krt12rtTA/tet-o-Cre/Ctnnb1 ), constitutive nuclear translocation of mutant beta-catenin ( loss of exon 3 ) leads to hyper proliferation of corneal epithelial cells ; in comparison of expression of beta-catenin mutant protein by migrating, periocular mesenchymal cells of Kera-rtTA/tet-o-Cre/Ctnnb1 caused eyelid malformation