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AHSA1 — POLDIP2

Text-mined interactions from Literome

Bhowmick et al., J Biol Chem 2001 : In summary, TGF-beta mediated p38MAPK activation is dependent on functional integrin beta(1), and p38MAPK activity is required but is not sufficient to induce EMT
Zhu et al., J Biol Chem 2002 (Anoxia) : Hypoxic neuronal death was also reduced by p38 inhibitors, by dominant interfering MKK3, and by a p53-antisense oligodeoxynucleotide and was increased by a constitutively active form of p38 and by an Mdm2-antisense oligodeoxynucleotide
Okuma et al., Exp Hematol 2002 (MAP Kinase Signaling System) : In contrast, both TNF alone and cytokine combination equivalently phosphorylated p38 in U937 cells, which was dissociated from differentiation, and a specific inhibitor of p38 ( SB203580 ) did not inhibit differentiation
Endo et al., J Recept Signal Transduct Res 2003 : Inhibition of VEGFR-2 blocked activation of extracellular regulated-kinase, p38 , Akt, and endothelial nitric oxide synthetase ( eNOS ) by VEGF, but did not inhibit p38 activation by the VEGFR-1-specific ligand, placental growth factor ( PIGF )
Shih et al., Food Chem Toxicol 2005 (Adenocarcinoma...) : In addition, the malvidin treatment significantly increased the p38 kinase expression and inhibited the ERK activity, and the effects of malvidin on caspase-3 activation were blocked , respectively, by the ERK and p38 inhibitors
Tsai et al., J Cell Biochem 2008 : Cilostazol significantly inhibited LPS induced activation of p38 , JNK, and nuclear factor-kappaB, and the respective inhibitors of p38 and JNK greatly reduced the level of LPS induced MCP-1 and MMP-9, suggesting the involvement of the p38 and JNK pathways
Nencioni et al., J Biol Chem 2009 : In infected Bcl-2 ( + ) cells, activated p38MAPK was found predominantly in the cytoplasm, colocalized with Bcl-2, and both Bcl-2 phosphorylation and virally induced apoptosis were diminished by specific inhibition of p38MAPK activity
Imai et al., J Periodontol 2009 (Halitosis...) : The activation of ERK1/2 and p38 induced by H2S was inhibited by the specific inhibitor of MAPK/ERK kinase ( [ MEK ] ; U0126 ) or p38 ( SB203580 )
Choi et al., Mol Pharmacol 2010 (Breast Neoplasms) : Collectively, capsaicin induced autophagy is regulated by p38 and ERK ; p38 controls autophagy at the sequestration step, whereas ERK controls autophagy at the maturation step, and that autophagy is involved in the retardation of cell death by blocking capsaicin induced ER stress mediated apoptosis in MCF-7 and MDA-MB-321 cells
Sparkenbaugh et al., Am J Physiol Gastrointest Liver Physiol 2012 : Hx-EL cotreatment caused an additive increase in phosphorylated p38 , and p38 inhibition attenuated cell death caused by this cotreatment
Mamik et al., PloS one 2012 : Furthermore, SHP2 overexpression resulted in higher SHP2 and p38 enzyme activity whereas p38 overexpression resulted in higher p38 but not SHP2 enzyme activity
Storm et al., PloS one 2013 : Through global transcriptional analysis and phosphorylation arrays, a strong ion flux dependent p38 MAPK response was detected and inhibition of p38 signaling delayed HAMLET induced death
Yang et al., Ann Hepatol 2013 (Cholelithiasis) : Expression of phosphorylated p38MAPK was upregulated by exogenous PGE2, and p38MAPK inhibitor reduced MUC2 and MUC5AC expression in HIBECs
Wagner et al., Digestion 1999 (Pancreatitis) : Pancreatic expression of p38 Map kinase and MAPK, SAPK and p38 regulation during pancreatic stress were investigated