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FOS — SRF

Pathways - manually collected, often from reviews:

• FastForward regulation: SRF → FOS (transcriptional regulation, unknown) Cooper et al., Genome Res 2007
  Evidence: DNABINDING
• FastForward regulation: SRF → FOS (transcriptional regulation, unknown) Chaudhary et al., Mol Endocrinol 1999*, Bernal-Mizrachi et al., J Biol Chem 2000*, White et al., Biochemistry 2000*, Drewett et al., J Biol Chem 2001*
  Evidence: DNABINDING
• FastForward regulation: SRF → FOS (transcriptional regulation, unknown) Boulden et al., Mol Cell Biol 1992*, Metz et al., Genes Dev 1991*, de Belle et al., Mol Cell Biol 1991*, Latinkic et al., Nucleic Acids Res 1991*, Rivera et al., Genes Dev 1990*
  Evidence: DNABINDING
• FastForward regulation: SRF → FOS (transcriptional regulation, unknown) Gille et al., Nature 1992*, Dalton et al., Cell 1992*, Treisman et al., EMBO J 1992*, Gualberto et al., Mol Cell Biol 1992*, Shaw et al., EMBO J 1992*
  Evidence: DNABINDING
• KEGG MAPK signaling pathway: Complex of ELK1-ELK4-SRF → FOS (gene expression, expression)

Text-mined interactions from Literome

Bruning et al., Proc Natl Acad Sci U S A 2000 : Thus Rsk-2 is required for growth factor stimulated expression of c-Fos and transcriptional activation of Elk-1 and the serum response factor , but not for activation of CREB or the mitogen activated protein kinase pathway in response to PDGF and IGF-1 stimulation
Hao et al., FEBS Lett 2006 : Mutants of ITK that lack kinase activity or a kinase domain can rescue cells lacking Tec family kinases for antigen receptor induced SRF activation, but not for NFAT, AP-1 or NFkappaB activation
Nagashima et al., J Biol Chem 2011 (Ataxia) : Mutation analysis indicated that the nuclear localization signal and both the N- and C-terminal regions of CRAG are critical for SRF dependent c-Fos activation