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CISH — CRK

Text-mined interactions from Literome

Deschesnes et al., Mol Biol Cell 2001 : In cells with deregulated c-Myc expression but not in control cells, cis-diamminedichloroplatinum induced p38 activity and typical features of apoptosis, including internucleosomal DNA degradation, induction of caspase activities, and both nuclear ( nuclear condensation and fragmentation ) and extranuclear ( cell blebbing ) morphological alterations
Bode et al., Biol Chem 2001 (Carcinoma, Hepatocellular) : We analyzed the role of MKK6/p38MAPK for IL-6 signal transduction and transcriptional activation of the suppressor of cytokine signaling (SOCS) 3 promoter
Nätzker et al., Biol Chem 2002 (Neuroblastoma) : In B104 cells, cis-4-methylsphingosine stimulated p38 mitogen activated protein kinase ( p38 MAPK ) and simultaneously inhibited extracellular signal regulated kinase ( ERK ), whereas sphingosine and sphingosine-1-phosphate only stimulated p38 MAPK without suppression of ERK
Frobøse et al., Mol Endocrinol 2006 : Suppressor of cytokine signaling (SOCS)-3 was shown to inhibit IL-1 induced transcription and activation of NFkappaB and the MAPKs JNK and p38 , but the mechanism is unknown
Zhao et al., J Biol Chem 2008 (MAP Kinase Signaling System) : To understand the mechanism of cis-elements regulated by p38alpha at post-transcriptional level, truncation of 3'-UTR and the full-length 3'-UTR with individual AUUUA motif mutation placed in gene reporter system was employed
Hernandez-Flores et al., BMC cancer 2011 (Uterine Cervical Neoplasms) : CIS induces important levels of senescence and phosphorylation of ERK1/2, p38 , p65/RELA, and I?Ba, and decreased the expression of anti-apoptotic protein Bcl-XL