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IAPP — JUN

Text-mined interactions from Literome

Zhang et al., J Mol Biol 2002 (Insulinoma) : Our results suggest strongly that human amylin induces apoptosis through stimulation of expression and activation of c-Jun , and that co-expression and dimerization of c-Jun and c-fos or ATF-2 may be important for activation of the downstream apoptotic process
Zhang et al., J Biol Chem 2003 (Insulinoma) : Furthermore, we showed that specific jnk1 antisense oligonucleotides, which suppress phospho-JNK1 expression, effectively decreased human amylin induced activation of c-Jun
Marusina et al., J Immunol 2008 : Overexpression of c-Jun or c-Fos in NK and T cells led to enhanced DAP10 promoter activity and DAP10 protein expression ... Taken together, our data indicate that Ap-1 is an important transcription factor for regulating DAP10 gene expression in human NK and T cells, and that Ap-1 plays a key role in the transactivation of DAP10 promoter following TCR stimulation