◀ Back to FAS
BCR — FAS
Text-mined interactions from Literome
Yoshida et al., Int Immunol 2000
(Immunologic Deficiency Syndromes) :
These results indicate that
BCR ligation activates an apoptotic signaling pathway distinct from Fas mediated apoptosis in WEHI-231 cells, and that BCR mediated apoptosis of WEHI-231 cells does not
require Fas or related molecules such as DR3, DR4 and DR5, as all of these death receptors require FADD/MORT1 and/or CrmA-sensitive caspases for induction of apoptosis
Huang et al., J Biol Chem 2013
(Leukemia, Myelogenous, Chronic, BCR-ABL Positive) :
We found previously that expression of the CML related
Bcr-abl oncoprotein in myeloid progenitor cells
increases expression of
Fas associated phosphatase 1 (Fap1)
Galibert et al., J Exp Med 1996
:
CD40 +
BCR stimulation of resting B cells with CD40 ligand transfected fibroblastic cells in the presence of cross linked anti-BCR monoclonal antibodies
resulted in the coexpression of CD38,
CD95 , and CPM
Wang et al., J Exp Med 1996
:
Moreover,
BCR cross linking in Lyn-/- B cells
suppressed Fas expression induced by costimulation with CD40 ligand and IL-4
Selleri et al., Blood 1998
(Leukemia, Myelogenous, Chronic, BCR-ABL Positive...) :
Fas mediated modulation of
Bcr/Abl in chronic myelogenous leukemia results in differential effects on apoptosis