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ATF4 — GDNF

Text-mined interactions from Literome

Ameri et al., Blood 2004 (Anoxia) : By applying RNAi, ATF-4 induction in anoxia was shown to be independent of HIF-1alpha, and desferrioxamine mesylate ( DFO ) and cobalt chloride induced HIF-1alpha but not ATF-4 or GADD153
Wek et al., Biochem Soc Trans 2006 : ATF4 also enhances the expression of additional transcription factors, ATF3 and CHOP ( CCAAT/enhancer binding protein homologous protein ) /GADD153 ( growth arrest and DNA-damage-inducible protein ), that assist in the regulation of genes involved in metabolism, the redox status of the cells and apoptosis
Hengstermann et al., Free Radic Biol Med 2008 : Activation of PERK and PERK dependent signaling by aqueous extracts of CS was demonstrated by ( i ) the inhibition of protein synthesis, ( ii ) the phosphorylation of PERK and its substrate eIF2alpha, ( iii ) the activation of ATF4 , and ( iv ) the expression of ATF4 dependent target genes chop, gadd34, BiP, and atf3