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ICAM1 — NM

Text-mined interactions from Literome

Vainer et al., Scand J Gastroenterol 2001 (Colitis, Ulcerative) : Impaired sensitivity to beta 2 integrin blocking in ICAM-1 mediated neutrophil migration in ulcerative colitis
Wang et al., J Biol Chem 2003 : Activation of SRC tyrosine kinases in turn leads to tyrosine phosphorylation of ezrin, as well as activation of p38, a kinase previously identified to be required for cytoskeletal changes induced by ICAM-1 ligation and for neutrophil migration along the EC surface
Dal Secco et al., Nitric Oxide 2006 : In conclusion, the results suggest that although during inflammation, ICAM-1 does not contribute to neutrophil migration , it is necessary for the down-modulatory effect of inflammation released NO on the adhesion and transmigration of neutrophils
Monson et al., Journal of inflammation (London, England) 2007 : P-selectin and the intercellular adhesion molecule (ICAM)-1 can mediate neutrophil-endothelial cell interactions, neutrophil migration , and the interactions of neutrophils with hepatocytes in the liver
Agace et al., Eur Respir J 1996 : E. coli enhanced the expression of intercellular adhesion molecule-1 ( ICAM-1 ) on urinary tract epithelial cells, and neutrophil migration across urinary tract epithelial layers in vitro was dependent on epithelial ICAM-1 and neutrophil Mac-1 ( CD11b/CD18 ) expression