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ICAM1 — SELP

Text-mined interactions from Literome

Folch et al., Ann Surg 1999 (Acute Disease...) : Pancreatitis induces increases in P-selectin expression in lung, whereas ICAM-1 is unchanged from baseline levels
Wang et al., Zhonghua Zheng Xing Shao Shang Wai Ke Za Zhi 1998 (Burns) : Under the influence of burn patient sera, the expression of P-selectin increased significantly at 1 hour ( P < 0.01 ) in hepatic sinusoidal endothelial cells but not in hepatocytes, and the expression of ICAM-1 increased significantly at 1 and 2 hour ( P < 0.01 ) in hepatocytes and hepatic sinusoidal endothelial cells
Zhou et al., World J Gastroenterol 2005 (Reperfusion Injury) : P-selectin increased significantly in hepatic sinusoidal endothelial cells and renal tubular epithelial cells 1 h after ischemia-reperfusion, and the expression of ICAM-1 was up-regulated in hepatic sinusoid and renal vessels after 6 h. CD1a ( + ) CD80 ( + ) DCs gradually increased in hepatic sinusoidal endothelium and renal tubules and interstitium 1 h after ischemia-reperfusion, and there was the most number of DCs in 24-h group
Sondén et al., J Investig Med 2006 (Inflammation) : Expression of P-selectin was enhanced 11-fold compared with controls, whereas expression of E-selectin and intercellular adhesion molecule 1 was enhanced 8-fold ( p < .05 ) and 1.5-fold ( p < .01 ), respectively
Yoshizaki et al., J Immunol 2010 (Fibrosis...) : This study suggests that L-selectin and ICAM-1 regulate Th2 and Th17 cell accumulation into the skin and lung, leading to the development of fibrosis, and that P-selectin , E-selectin, and PSGL-1 regulate Th1 cell infiltration, resulting in the inhibition of fibrosis
Lin et al., J Pharm Pharmacol 2013 (Body Weight...) : Eugenosedin-A reduced the upregulation of P-selectin , ICAM-1, ICAM-2, ICAM-3, VCAM, PECAM in platelets and inhibited E-selectin, ICAM-1 , ICAM-2, ICAM-3, VCAM and PECAM protein levels in the aorta ... Eugenosedin-A reduced the upregulation of P-selectin , ICAM-1, ICAM-2, ICAM-3, VCAM, PECAM in platelets and inhibited E-selectin, ICAM-1, ICAM-2, ICAM-3 , VCAM and PECAM protein levels in the aorta
Cockerill et al., Lab Invest 1994 (Trisomy) : C11STH expresses von Willebrand factor, P-selectin , and scavenger receptor to an extent similar to passaged HUVECs and can be induced to express E-selectin, VCAM, and ICAM with TNF-alpha
Delneste et al., Clin Exp Immunol 1994 : Histamine did not modulate the expression of intercellular adhesion molecule-1 ( ICAM-1 ), vascular cell adhesion molecule-1 ( VCAM-1 ) and E-selectin, but induced a transient expression of P-selectin as previously reported
Shen et al., J Virol 1997 : TNF-alpha significantly enhanced WNV induced increases in E-selectin, P-selectin , ICAM-1, and VCAM-1 expression, while IFN-gamma enhanced WNV induced ICAM-1 expression
Nishio et al., Am J Respir Crit Care Med 1998 (Hyperoxia) : We found that P-selectin expression was sparsely enhanced only in arterioles, whereas ICAM-1 was significantly induced in both venules and capillaries