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UCSC Genome Browser Gene Interaction Graph
Gene interactions and pathways from curated databases and text-mining

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IL10 — SELE

Text-mined interactions from Literome

Cockerill et al., Circulation 2001 (Acute Disease...) : Elevation of plasma high-density lipoprotein concentration reduces interleukin-1 induced expression of E-selectin in an in vivo model of acute inflammation
Landis et al., Arthritis Rheum 2002 : Human peripheral blood monocytes were differentiated for 1-7 days in vitro and examined with respect to 1 ) uptake of MSU crystals, 2 ) expression of macrophage, dendritic cell, and activation markers, 3 ) secretion of tumor necrosis factor alpha (TNFalpha), interleukin 1beta (IL-1beta), IL-6, and IL-10 , 4 ) activation of endothelial E-selectin expression, and 5 ) enhancement of secondary neutrophil recruitment by endothelial cells
Wereszczynska-Siemiatkowska et al., Pancreas 2003 (Abdominal Pain...) : At day 10, the E-selectin level in severe AP was still higher than that in mild AP, and the IL-10 concentration increased again
Furbert-Harris et al., Cell Mol Biol Incl Cyto Enzymol 2003 (Neoplasm Metastasis...) : TNF-alpha and IL-10 enhanced ELAM-1 on DU 145, but EosA 24 hr supematants failed to do so
Mitoma et al., Gastroenterology 2005 (Crohn Disease) : Both anti-TNF agents induced E-selectin expression, but only infliximab induced interleukin-10 production, apoptosis, and G0/G1 cell cycle arrest
Leeuwenberg et al., Eur J Immunol 1991 (Leukemia, Myeloid) : ELAM-1 expression can selectively be induced on human umbilical vein endothelial cells ( HUVEC ) by tumor necrosis factor, interleukin 1 and lipopolysaccharide
Lewis et al., Mol Cell Biol 1994 : In addition, an essential role for cooperative interaction between the two NF-kappa B complexes is shown by the requirement for both NF-kappa B sites to mediate E-selectin promoter activation by interleukin-1 and p50/p65 expression
Weston et al., Transpl Immunol 1995 (Kidney Failure, Chronic) : E-Selectin is a 115-kDa cell surface glycoprotein transiently expressed on vascular endothelium in response to interleukin-1 and tumour necrosis factor-alpha with a peak in expression at four hours
Vora et al., J Exp Med 1996 : Interleukin-10 induces E-selectin on small and large blood vessel endothelial cells ... These studies demonstrate that IL-10 induces the expression of E-selectin on both HDMEC and HUVEC and that the level of expression of HDMEC is comparable with that induced by IL-1 beta and tumor necrosis factor-alpha ... The induction of the expression of E-selectin by IL-10 on HDMEC may provide additional insights into the pathogenic mechanism of neutrophil accumulation at the site of inflammation in inflammatory skin diseases
Ray et al., Biochem J 1997 : Induction of the E-selectin promoter by interleukin 1 and tumour necrosis factor alpha, and inhibition by glucocorticoids
Miller et al., J Leukoc Biol 1998 : Utilizing neutralizing antibodies, we show that CD40L mediated tissue factor and thrombomodulin modulation, as well as E-selectin and VCAM-1 upregulation, is independent of tumor necrosis factor alpha, interleukin-1alpha , or interleukin-1beta production
Friedrichs et al., Arterioscler Thromb Vasc Biol 1998 : Inhibition of tumor necrosis factor-alpha- and interleukin-1 induced endothelial E-selectin expression by thiol modifying agents