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AKT1 — CAT

Text-mined interactions from Literome

Ushio-Fukai et al., J Biol Chem 1999 (Hypertrophy) : Furthermore, diphenylene iodonium, an inhibitor of flavin containing oxidases, or overexpression of catalase to block angiotensin II-induced intracellular H ( 2 ) O ( 2 ) production significantly inhibits angiotensin II-induced Akt/PKB phosphorylation, indicating a role for ROS in agonist induced Akt/PKB activation
Jang et al., Cell Signal 2005 : Interestingly, there was PI3K dependent activation of AKT , p70S6K, JNKs, and NF-kappaB in response to catalase
Venkatesan et al., J Cell Physiol 2007 : Furthermore, expression of dominant negative Akt and constitutively active FoxO1 increased catalase transcription, respectively
Yi et al., Nutrition 2010 (Hypertension...) : No differences in superoxide dismutase and glutathione peroxidase activities were observed between SHRSPs and SHRs. Clofibrate treatment significantly upregulated renal HMGCS2 expressions, inhibited phosphorylation of Akt , and decreased malondialdehyde levels and catalase activities in SHRSP kidney tissues ( P < 0.05 )
Saunders et al., Free Radic Biol Med 2010 (Ovarian Neoplasms) : Akt phosphorylation was also inhibited by PEG-catalase and apocynin
Li et al., PloS one 2013 : Furthermore, we found that the presence of NAC, as well as the overexpression of MnSOD, could almost completely abolish the activation of Akt , extracellular-signal regulated kinase ( Erk ) 1/2, c-Jun N-terminal kinase (JNK), and p38, although only JNK and p38 were affected by overexpression of catalase