◀ Back to VEGFA

MRXS5 — VEGFA

Text-mined interactions from Literome

Bamba et al., Biochem Biophys Res Commun 2000 (Colonic Neoplasms) : These results suggest that PGs dramatically increased VEGF production by activated macrophages through specific PGE receptor and PPARgamma mediated processes and that PGs may thereby promote tumor growth through VEGF production
Bamba et al., J Exp Clin Cancer Res 2000 (Colonic Neoplasms) : These findings suggest that PGs produced by COX-2 expressing macrophages induce VEGF production by macrophages, but not by cancer cells, in an autocrine fashion
Amano et al., Jpn J Pharmacol 2001 : We previously reported that endogenous prostaglandins (PGs) may increase cAMP facilitated angiogenesis through the induction of vascular endothelial growth factor ( VEGF ) in rat sponge implantation models
Bamba et al., Aliment Pharmacol Ther 2003 : We previously reported that prostaglandins (PGs) dramatically increased vascular endothelial growth factor ( VEGF ), a known angiogenic factor and a vascular permeable factor, by activated macrophages through specific PGE receptor and peroxisome proliferator activated receptor gamma ( PPARgamma, a nuclear receptor of PG ) mediated process
Cheng et al., Int J Oncol 2004 (Carcinoma, Hepatocellular...) : The effects of PGs on VEGF expression were also investigated
Harada et al., J Clin Invest 1994 : Stimulation of VEGF expression by PGs and its suppression by glucocorticoids, which, respectively, stimulate and suppress bone formation, strongly implicate the involvement of VEGF in bone metabolism
Takahashi et al., J Clin Gastroenterol 1998 (Stomach Ulcer) : Our data indicate that PGs stimulate VEGF expression in gastric fibroblasts