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BCL2 — SLC17A5

Text-mined interactions from Literome

Selzner et al., J Hepatol 2002 (Reperfusion Injury) : Bcl-2 overexpression resulted in significant decreased AST levels after ischemic injury, and complete inhibition of apoptosis
Lee et al., Food Chem Toxicol 2011 (MPTP Poisoning) : Furthermore, AST is able to enhance the expression of Bcl-2 protein but reduce the expression of a-synuclein and Bax, and suppress the cleavage of caspase-3 ... Our results suggest that the protective effects of AST on MPP+ induced apoptosis may be due to its anti-oxidative properties and anti-apoptotic activity via induction of expression of superoxide dismutase ( SOD ) and catalase and regulating the expression of Bcl-2 and Bax