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FST — IGF1

Text-mined interactions from Literome

Mihm et al., Domest Anim Endocrinol 2002 : In order to elucidate the mechanisms underlying DF survival or SF atresia, this review aims to ( i ) describe follicular changes in the local production and regulation of members of the inhibin family of proteins and the insulin-like growth factor (IGF) system in relation to FSH deprivation leading to DF selection, and ( ii ) develop a model for DF selection outlining the putative involvement of inhibins, activin and follistatin on the one hand, and bioavailable IGFs regulated by IGF binding proteins ( IGFBPs ) and IGFBP proteases on the other hand
Glister et al., Reproduction 2004 : This was reinforced by functional experiments on granulosa cells which showed that BMP-4, -6 and -7 promoted cellular accumulation of phosphorylated Smad-1 but not Smad-2 and enhanced ` basal ' and IGF stimulated secretion of oestradiol ( E2 ), inhibin-A, activin-A and follistatin (FS)