Gene interactions and pathways from curated databases and text-mining

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FOS — WAS

Text-mined interactions from Literome

Allen et al., Neuropharmacology 2003 : Results showed that naloxone inhibited THC induced Fos immunoreactivity in several key brain regions including the ventral tegmental area, ventromedial and dorsomedial hypothalamus, central caudate-putamen and ventrolateral periaqueductal grey ... Conversely, naloxone and THC had an additive effect on Fos immunoreactivity in the central nucleus of the amygdala, the bed nucleus of the stria terminalis ( lateral division ), the insular cortex, and the paraventricular nucleus of the thalamus
Van Sickle et al., Am J Physiol Gastrointest Liver Physiol 2003 (Vomiting) : At the level of the obex, THC reduced Fos expression in the area postrema and the dorsal subnucleus of the NTS
Boucher et al., Neuroscience 2007 : In the lateral septum, THC selectively increased c-Fos expression in Nrg1 HET mice with no corresponding effect being observed in WT mice ... In addition, THC promoted a greater increase in c-Fos expression in Nrg1 HET mice than WT mice in the central nucleus of the amygdala, the bed nucleus of the stria terminalis and the paraventricular nucleus of the hypothalamus
Goebel-Stengel et al., Brain Res 2011 : WAS induced Fos expression in 35 % of NUCB2/nesfatin-1-immunoreactive neurons in the PVN, 50 % in the LC, 54 % in the rostral raphe pallidus, 58 % in the VLM, 39 % in the middle part of the nucleus of the solitary tract (NTS) and 33 % in the caudal NTS
Miyamoto et al., Brain Res 1996 : SCH-23390, a selective dopamine D1 receptor antagonist, at 0.32 mg/kg produced a significant block of the effects of THC on Fos expression in the striatum and the nucleus accumbens ... Administration of 100 mg/kg ( - ) -sulpiride, a selective dopamine D2 receptor antagonist, failed to block the effect of THC on Fos expression in both the striatum and the nucleus accumbens ... These findings indicate that THC induces the expression of Fos protein and that this expression is mediated at least by dopamine D1 receptors
Porcella et al., Eur J Neurosci 1998 : In the cingulate cortex and caudate-putamen, delta9-THC significantly increased AP-1 DNA binding activity only at the highest dose used ( 57 and 71 %, respectively )