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ADORA3 — EPHB2

Text-mined interactions from Literome

Schulte et al., Mol Pharmacol 2002 : Furthermore the ERK1/2 phosphorylation was sensitive to the PI3K inhibitors wortmannin and LY294002 ( 2- ( 4-morpholinyl ) -8-phenyl-1 ( 4H ) -benzopyran-4-one hydrochloride ) and the MEK inhibitor PD98059 ( 2'-amino-3'-methoxyflavone ), whereas chelation of Ca ( 2+ ) with 1,2-bis ( 2-aminophenoxy ) ethane-N, N,N ', N'-tetraacetic acid tetrakis ( acetoxymethyl ester ) and long-term treatment with phorboldibutyrate did not decrease the adenosine A(3) receptor mediated ERK1/2 phosphorylation
Schulte et al., Exp Cell Res 2003 : Thus, stimulation of adenosine A ( 2B ) receptors activates both ERK1/2 and p38 via cAMP, but the downstream pathways are markedly different
Hammarberg et al., Biochem Pharmacol 2004 : Furthermore, we identified the serine 727 site of signal transducer and activator of transcription STAT3 as a probable downstream target of ERK1/2, and thereby provide evidence that adenosine A(3) receptor mediated ERK1/2 activation has functional consequences
Button et al., Exp Physiol 2005 : Phosphatidylinositol 3-kinase and ERK1/2 are not involved in adenosine A1 , A2A or A3 receptor mediated preconditioning in rat ventricle strips
Nakata et al., Biochem Biophys Res Commun 2007 : These neurite outgrowth and ERK1/2 activation were significantly inhibited by PD98059, an inhibitor of mitogen activated protein kinase kinase, as well as by activation of endogenous adenosine A2A receptors