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CALM3 — EPHB2

Text-mined interactions from Literome

Luo et al., J Neurochem 1999 (Neuroblastoma) : Thus, TGFbeta1 affects ERK dependent cell proliferation and ERK independent N-CAM expression in B104 cells
Lee et al., Brain Res Mol Brain Res 2000 : This finding indicates that the calmodulin dependent activation of ERK and p38 kinase is involved in calcium induced c-fos expression
Rosengart et al., Shock 2000 : Interactions of calcium/calmodulin dependent protein kinases ( CaMK ) and extracellular regulated kinase (ERK) in monocyte adherence and TNFalpha production
Stocco et al., J Biol Chem 2002 : A calcium/calmodulin dependent activation of ERK1/2 mediates JunD phosphorylation and induction of nur77 and 20alpha-hsd genes by prostaglandin F2alpha in ovarian cells
Ferrer et al., J Neural Transm 2001 (Alzheimer Disease...) : Calcium/calmodulin dependent kinase II ( alpha- and beta-CaM kinase II ), and phosphorylated mitogen activated extracellular signal regulated protein kinase ( MAPK/ERK-P ), phosphorylated protein kinase of 38 kDa ( p38-P ) and phosphorylated stress activated protein kinase ( SAPK/JNK-P ) expression have been examined in Alzheimer disease (AD), Pick's disease (PiD), progressive supranuclear palsy ( PSP ) and corticobasal degeneration (CBD)
Verploegen et al., Eur J Biochem 2002 (Calcium Signaling) : Inhibition of calmodulin using a calmodulin antagonist N- ( 6-aminohexyl ) -5-chloro-1-naphthalenesulfonamide ( W7 ), did not effect N-formyl-methionyl-leucyl-phenylalanine ( fMLP ) induced ERK , p38 MAPK or GSK-3alpha phosphorylation, but attenuated fMLP induced PKB phosphorylation
Arnette et al., J Biol Chem 2003 : In particular, calmodulin antagonists, FK506, and cyclosporin, immunosuppressants that inhibit the calcium dependent phosphatase calcineurin, suppress ERK1/2 activation by both glucose and GLP-1
Sgambato et al., J Neurochem 2003 : This effect depended on the activation of NMDA receptors but was independent of both calcium/calmodulin dependent protein kinases ( CaMK ) and extracellular regulated kinase (ERK)
Kameshita et al., J Biochem Biophys Methods 2004 : One of the antibodies obtained, KI98, detected a variety of purified Ser/Thr protein kinases, such as calmodulin dependent protein kinase II ( CaM-kinase II ), calmodulin dependent protein kinase IV ( CaM-kinase IV ), cAMP dependent protein kinase, protein kinase C, and Erk2
Xie et al., Neuromolecular Med 2004 (Alzheimer Disease...) : Interestingly, acute application of Abeta or APP overexpression inhibits activity dependent regulation of several protein kinase pathways that require Ca2+ influx via NMDA receptors for activation, including Ca2+/calmodulin dependent protein kinase II, protein kinase A, and extracellular regulated kinases (Erk)
Roberson et al., Mol Endocrinol 2005 : Inhibition of Cam ( using a dominant negative ) was sufficient to block GnRH induced ERK but not c-Jun N-terminal kinase activity activation
Shinkai et al., Am J Physiol Lung Cell Mol Physiol 2006 : At 120 min and 6 h, CAM increased phospho-ERK1/2 ( pERK ) but not phospho-p38 or phospho-JNK
Méndez-Samperio et al., J Infect 2006 (Calcium Signaling...) : Activation of ERK1/2 and TNF-alpha production are mediated by calcium/calmodulin , and PKA signaling pathways during Mycobacterium bovis infection
Shinkai et al., Antimicrob Agents Chemother 2006 : CAM also inhibited ERK1/2 phosphorylation over the first 90 min of exposure ( P < 0.05 for 30 min, P < 0.0001 for 60 min, and P < 0.01 for 90 min ) and decreased the ratio of phosphorylated ERK1/2 ( pERK1/2 ) to total ERK1/2 ( tERK1/2 ) ( P < 0.0001 )
Norum et al., FEBS Lett 2007 (Calcium Signaling...) : However, serotonin stimulated phosphorylation of endogenous and recombinant Ras-GRF1, increased [ Ca ( 2+ ) ] ( i ) and caused Ca ( 2+ ) - and calmodulin dependent ERK1/2 phosphorylation
Zhao et al., J Neurosci Res 2008 (Brain Ischemia...) : Protein phosphatase 2A-negative regulation of the protective signaling pathway of Ca2+/CaM dependent ERK activation in cerebral ischemia
Cammarota et al., Proc Natl Acad Sci U S A 2008 (Amnesia) : IA training is followed by an increase of the phosphorylation of calcium and calmodulin dependent protein kinase II ( CaMKII ) and ERK2 in CA1 but only an increase of the phosphorylation of ERK2 in BLA
Labasque et al., Mol Biol Cell 2008 (MAP Kinase Signaling System) : Expression of the CaM mutant also prevented receptor mediated ERK1,2 phosphorylation in cultured cortical neurons and choroid plexus epithelial cells that endogenously express 5-HT(2C) receptors
Zheng et al., J Neurosci Res 2009 : Moreover, we found that inhibition of calmodulin (CaM) by W13 blocked both Arc transcription and ERK activation, revealing a Ca ( 2+ ) -independent function of CaM
Cipolletta et al., Endocrinology 2010 : Calcium dependent signaling through calcium/calmodulin dependent kinase II ( CaMKII ) and ERK1/2 activation plays an important role in the regulation of VSMC proliferation by agents such as alpha-adrenergic receptor agonists
Kitao et al., Neurochem Int 2010 (Calcium Signaling) : Inhibitors of intracellular Ca ( 2+ ) -dependent proteins such as calpain and calmodulin blocked SNP induced ERK phosphorylation and decrease in cell viability
Furuya et al., Journal of inflammation (London, England) 2010 : CAM and M-4 also suppressed phosphorylation of MAPKs, ERK and p38 MAPK, but not JNK, which are increased LPS stimulation
Abbott et al., J Appl Physiol 2011 (Calcium Signaling) : Glucose uptake rates were blunted in caffeine treated DN mice ( P < 0.05 ). The DN transgene resulted in a greater use of intramuscular triglycerides as a fuel source during muscle contraction. The DN transgene did not alter caffeine- or contraction mediated changes in the phosphorylation of Ca ( 2+ ) /calmodulin dependent protein kinase I or ERK1/2 ( P > 0.05 )
Moncada et al., Proc Natl Acad Sci U S A 2011 : Moreover, activation of the glutamatergic NMDA receptors is required for setting the learning tags, and this machinery further required a-Ca ( 2+ ) /calmodulin dependent protein kinase II and PKA but not ERK1/2 activity
Lucki et al., Mol Cell Endocrinol 2012 (Calcium Signaling...) : S1P induced StAR mRNA expression requires Ga ( i ) signaling, phospholipase C (PLC), Ca ( 2+ ) /calmodulin dependent kinase II ( CamKII ), and ERK1/2 activation
Guo et al., Exp Cell Res 2012 : Also, ERK and CREB phosphorylation was not mediated by protein kinase A (PKA) or calmodulin kinase ( CaMK )
Kramarenko et al., Cancer management and research 2012 : Inhibitor studies suggest that BK-induced ERK activation requires phospholipase C and protein kinase C activities, and is Ca ( 2+ ) /calmodulin dependent
Egea et al., J Neurochem 1998 (Ion Channel Gating) : These data suggest that calmodulin mediates ERK activation induced by increases in intracellular Ca2+ concentration in PC12 cells by a mechanism that seems to be independent of Trk A and EGFR activation
Bosch et al., J Biol Chem 1998 : Because calmodulin is essential for quiescent cells to enter cell cycle, the role of calmodulin on ERK2 activation was studied in cultured fibroblasts
Murasawa et al., Hypertension 1998 : Calmodulin inhibitors ( W7 and calmidazolium ) and tyrosine kinase inhibitors ( genistein and ST638 ) completely blocked ERK activation by Ang II and A23187 ... These findings demonstrate that in cardiac fibroblasts, Ang II induced Ras/ERK activation is dominantly regulated by Gq-coupled Ca2+/calmodulin signaling and that Pyk2 plays an important role in the signal transmission for efficient activation of the Ang II induced Ras/ERK pathway