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JUN — PDGFB

Text-mined interactions from Literome

Franchimont et al., J Biol Chem 1999 : Electrophoretic mobility shift assay revealed AP-1 and CRE nuclear protein complexes that were enhanced by PDGF BB
Zhan et al., Arterioscler Thromb Vasc Biol 2002 : Our results provide the first evidence that dominant negative c-Jun inhibits PDGF-BB induced vascular SMC proliferation by preventing the downregulation of p27 ( Kip1 ), thereby supporting the important role of c-Jun in vascular SMC proliferation
Bhanoori et al., Oncogene 2003 : Thiol alkylation inhibited PDGF-BB induced expression of the Fos and Jun family proteins and AP-1 activity in VSMC
He et al., Am J Physiol Heart Circ Physiol 2003 : Furthermore, decreasing Ref-1 protein by treatment of SMCs with Ref-1 AODN, or by immunodepletion of Ref-1 from nuclear extracts, inhibited PDGF-BB induced activator protein-1 (AP-1) DNA binding activity
Kagami et al., J Cell Physiol 2004 (Glomerulonephritis) : PDGF-BB enhanced not only gel contraction but ERK phosphorylation and AP-1 activity by MCs ... Marked inhibitory effects on PDGF-BB induced gel contraction and ERK/AP-1 activity were observed in the presence of either function blocking anti-alpha1- or anti-beta1-integrin antibody or U0126
Chiang et al., Life Sci 2007 (Melanoma) : PDGF-BB induced phosphorylation of PDGF receptor beta ( PDGFR-beta ), extracellular signal regulated kinase 1/2 ( ERK1/2 ), p38, and c-Jun N-terminal kinase (JNK) was attenuated by lycopene in a concentration dependent manner, whereas the total expression of each protein was not affected
Rovida et al., J Hepatol 2008 : ERK5 silencing inhibited PDGF-BB induced cell proliferation, and expression and activation of c-Jun
Sabuda-Widemann et al., Nephrol Dial Transplant 2009 : MPA treatment did not affect PDGF-BB induced AP-1 activity determined after 1 h and 2 h
Seidel et al., Schweiz Med Wochenschr 2010 : PDGF-BB and TNF-a activated AP-1 , which was also inhibited by DMF