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IL8 — SELE
Text-mined interactions from Literome
Cockerill et al., Circulation 2001
(Acute Disease...) :
Elevation of plasma high-density lipoprotein concentration reduces
interleukin-1 induced expression of
E-selectin in an in vivo model of acute inflammation
Lefranc et al., Clin Cancer Res 2004
(Brain Neoplasms...) :
Gastrin significantly ( but only transiently ) decreased the level of expression of E-selectin, but not P-selectin, whereas
IL-8 increased the expression of
E-selectin
Leeuwenberg et al., Eur J Immunol 1991
(Leukemia, Myeloid) :
ELAM-1 expression can selectively be
induced on human umbilical vein endothelial cells ( HUVEC ) by tumor necrosis factor,
interleukin 1 and lipopolysaccharide
Lewis et al., Mol Cell Biol 1994
:
In addition, an essential role for cooperative interaction between the two NF-kappa B complexes is shown by the requirement for both NF-kappa B sites to mediate
E-selectin promoter
activation by
interleukin-1 and p50/p65 expression
Weston et al., Transpl Immunol 1995
(Kidney Failure, Chronic) :
E-Selectin is a 115-kDa cell surface glycoprotein transiently expressed on vascular endothelium in
response to
interleukin-1 and tumour necrosis factor-alpha with a peak in expression at four hours
Bryant et al., Infect Immun 1996
:
PLC strongly
induced expression of
ELAM-1 , ICAM-1, and IL-8, while PFO stimulated early ICAM-1 expression but did not promote ELAM-1 expression or
IL-8 synthesis
Del Papa et al., Arthritis Rheum 1996
(Wegener Granulomatosis) :
We found that AECA IgG from WG patients do not display any significant cytotoxicity but are able to up-regulate the expression of
E-selectin , intercellular adhesion molecule 1 and vascular cell adhesion molecule 1 and to
induce the secretion of IL-1 beta, IL-6,
IL-8 , and MCP-1
Ray et al., Biochem J 1997
:
Induction of the
E-selectin promoter by
interleukin 1 and tumour necrosis factor alpha, and inhibition by glucocorticoids
Miller et al., J Leukoc Biol 1998
:
Utilizing neutralizing antibodies, we show that CD40L mediated tissue factor and thrombomodulin modulation, as well as
E-selectin and VCAM-1 upregulation, is
independent of tumor necrosis factor alpha,
interleukin-1alpha , or interleukin-1beta production
Friedrichs et al., Arterioscler Thromb Vasc Biol 1998
:
Inhibition of tumor necrosis factor-alpha- and
interleukin-1 induced endothelial
E-selectin expression by thiol modifying agents