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EPHB2 — FGFR4

Text-mined interactions from Literome

Gaubert et al., J Biol Chem 2001 : The expression of a dominant negative FGFR1 did not reduce ERK-1/2 activation by the HMW FGF-2, suggesting that ERK activation does not require FGFR activity
Cailliau et al., Biochim Biophys Acta 2001 : In contrast, FGFR4 stimulation by FGF2 induced an early transient activation of ERK2 and no meiosis reinitiation
Kontaridis et al., Mol Cell Biol 2002 : We propose that SHP-2 plays a pivotal role in FGFR signaling in myoblasts via both Erk dependent and Erk independent pathways
Li et al., J Cell Sci 2004 : FRS2 dependent SRC activation is required for fibroblast growth factor receptor induced phosphorylation of Sprouty and suppression of ERK activity
Lao et al., J Biol Chem 2007 : Direct binding of PP2A to Sprouty2 and phosphorylation changes are a prerequisite for ERK inhibition downstream of fibroblast growth factor receptor stimulation
Vincent et al., Osteoarthritis Cartilage 2007 (Mechanotransduction, Cellular) : The loading response was assessed by the activation of ERK , in the presence or absence of a specific FGFR inhibitor
Poliakov et al., J Cell Biol 2008 (MAP Kinase Signaling System) : In addition, FGFR1 signaling inhibits further phosphorylation of EphB2 upon stimulation with ephrinB1, and we show that this involves a requirement for the mitogen activated protein kinase ( MAPK ) pathway
Ono et al., Hum Mol Genet 2013 : The Ras-GTPase activity of neurofibromin restrains ERK dependent FGFR signaling during endochondral bone formation ... We provide here evidence that neurofibromin, via its Ras guanosine triphosphatase -activating activity, controls ERK1/2 dependent fibroblast growth factor receptor ( FGFR ) signaling in chondrocytes