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IL1R1 — TLR7
Text-mined interactions from Literome
Cheung et al., J Immunol 2005
:
However, AcPL is dispensable for the activation or
inhibition of
IL-1R and the various
TLR signals that we have examined
Zhang et al., J Immunol 2009
(Multiple Sclerosis, Relapsing-Remitting) :
We found that IFN-beta1a in vitro treatment of human monocyte derived DCs
induced the expression of
TLR7 and the members of its downstream signaling pathway, including MyD88, IL-1R associated kinase 4, and TNF receptor associated factor 6, while it inhibited the expression of
IL-1R ... Using small interfering RNA TLR7 gene silencing, we confirmed that IFN-beta1a induced changes in MyD88, IL-1R associated kinase 4, and
IL-1R expression were
dependent on
TLR7