◀ Back to NOS1

AVP — NOS1

Text-mined interactions from Literome

Martin et al., American journal of physiology. Renal physiology 2002 : Thus AVP specifically increases nNOS expression levels in the renal outer medulla and papilla ... Because nNOS is specifically expressed in principal cells of the collecting duct system, the stimulation of nNOS expression by AVP may participate in the control of water reabsorption
Ventura et al., Braz J Med Biol Res 2002 : The central inhibition of NOS by L-NAME caused a decrease in plasma AVP and no change in plasma OT or ANP levels after BVE
Fan et al., Life Sci 2007 (Cardiomyopathies...) : AVP also increased NOS activity, protein and mRNA levels of iNOS in a coincident manner ... In addition, it was found that NF-kappaB was activated by AVP , and that PDTC could inhibit NO production, NOS activity, protein and mRNA levels of iNOS stimulated by AVP in a dose dependent manner
Dewachter et al., Shock 2010 (Anaphylaxis) : Separately, aortic rings removed during early versus late anaphylaxis were contracted by AVP +/- pretreatment with N ( G ) -nitro-L-arginine methyl ester ( L-NAME ) ( 10 M ), a NOS inhibitor
Vega et al., Am J Physiol Regul Integr Comp Physiol 2010 (Hyperprolactinemia) : Here, we investigate whether the activation of neuronal nitric oxide synthase (nNOS) in the hypothalamo-neurohypophyseal system mediates the effect of PRL on OXT and AVP release and whether these effects operate in males
Chiodera et al., Neuroreport 1994 (Hypoglycemia) : Insulin induced hypoglycaemia, however, enhanced plasma AVP and OT levels by two-fold in the absence of L-NAME and by four-fold in the presence of the NO synthase inhibitor (NOS)
Yamamoto et al., Hypertension 1997 : We investigated the effects of arginine vasopressin (AVP) on nitric oxide ( NO ) synthase activity in cardiac myocytes by measuring the production of nitrite, a stable metabolite of NO, and the expression of inducible NO synthase (iNOS) mRNA and protein
Raber et al., Brain research. Brain research reviews 1998 (Disease Models, Animal...) : The IFNalpha stimulated release of AVP and CRH appears to be mediated by cyclic GMP production, and this release by IFNalpha and IL-2 may be mediated in part by activation of constitutive nitric oxide synthase