Chacko et al., BMC cancer 2010
(Carcinoma, Non-Small-Cell Lung...) :
Expression of VDAC1 is required for full processing and activation of caspase-8 and supports a role for mitochondria in regulating apoptosis signaling via the death receptor pathway
Keinan et al., Mol Cell Biol 2010
:
Finally, a caspase inhibitor had no effect on VDAC oligomerization and cytochrome c release
Wang et al., Food Chem Toxicol 2013
:
Pretreatment with selenium partially blocked Cd-induced ROS generation, inhibited Cd induced mitochondrial membrane potential collapse, prevented cytochrome c release, inhibited caspase activation and changed the level of VDAC , Bcl-2 and Bax