Gene interactions and pathways from curated databases and text-mining

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SETD2 — TP53

Protein-Protein interactions - manually collected from original source literature:

Studies that report less than 10 interactions are marked with *

Text-mined interactions from Literome

Choi et al., J Biochem Mol Biol 2003 (Cell Transformation, Neoplastic...) : It was recently reported that HIF-1alpha binds a co-activator of the AP-1 transcription factor, Jab-1, which inhibits the p53 dependent degradation of HIF-1 and enhances the transcriptional activity of HIF-1 and the subsequent VEGF expression under hypoxic conditions
Schmid et al., Biochem J 2004 (Carcinoma...) : In addition, low p53 expression repressed HIF-1 transactivation without affecting HIF-1alpha protein amount ... We conclude that low p53 expression attenuates HIF-1 transactivation by competing for p300, whereas high p53 expression destroys the HIF-1alpha protein and thereby eliminates HIF-1 reporter activity
Sohda et al., Int J Cancer 2004 (Esophageal Neoplasms...) : Previous reports have suggested that the regulation of p53 and p21 is HIF-1 dependent
Vleugel et al., Hum Pathol 2006 (Breast Neoplasms...) : Transcriptional activity of HIF-1 may be repressed by p53 through competition for transcriptional coactivators such as p300 ... This underlines the importance of p300 levels and p53 accumulation in the HIF-1 regulated response toward hypoxia
Hubert et al., J Cell Sci 2006 (Carcinoma, Hepatocellular...) : Ectopic overexpression of p53 also led to an inhibition of HIF-1 activity
Sano et al., Nature 2007 (Cardiac Output, Low...) : p53 induced inhibition of Hif-1 causes cardiac dysfunction during pressure overload
Sano et al., Nihon rinsho. Japanese journal of clinical medicine 2008 (Cardiomegaly...) : Cardiac angiogenesis played an important role in the maintenance of cardiac function as well as the development of cardiac hypertrophy induced by pressure-overload, and upregulated p53 induced the transition from cardiac hypertrophy to heart failure through the suppression of hypoxia inducible factor-1(HIF-1) , which regulates angiogenesis in the hypertrophied heart
Oda et al., PloS one 2008 : MIF regulates HIF-1 activity in a p53 dependent manner
Xie et al., Cell Signal 2008 : Here we show that SETD2 could interact with p53 and selectively regulate the transcription factor activity of p53
Hiwatashi et al., Anticancer Res 2009 (Anoxia) : Moreover, the p53 suppression induced by HIF-1a may lead to malignant potential in RAGE transfected Cos7 cells
Yoshioka et al., J Surg Res 2012 (Neovascularization, Pathologic...) : During hypoxia, p53 is stabilized by interaction with hypoxia-inducible factor-1 (HIF-1) ... Here, we showed that expression of wild-type p53, but not null or mutated p53 , significantly suppressed HIF-1 activity and production of VEGF, which mostly depends on the HIF-1ß protein level
de Lange et al., Oncogene 2012 (Anoxia...) : Also Topotecan, alone or in combination with Nutlin-3, reduced HIF-1a protein levels, suggesting that a certain level of DNA damage response is required for p53 mediated downregulation of HIF-1a
Kojima et al., Blood 2011 (Leukemia, Myeloid, Acute) : p53 activation by Nutlin-3a was not cytotoxic to stromal cells, but reduced CXCL12 mRNA levels and secretion of CXCL12 partially through p53 mediated HIF-1a down-regulation
Park et al., Mol Carcinog 2012 (Colonic Neoplasms...) : HFD feeding increased tumor tissue levels of Ki67, cyclin A, cyclin D1, CDK2, Bcl-xL, and Bcl-2 ; reduced p53 levels and TUNEL positive apoptotic cells ; increased the levels of CD45, CD68, CD31, VEGF, P-VEGF receptor-2, iNOS, and COX-2 as well as hemoglobin content ; and increased the levels of HIF-1a , P-STAT3-Y705, P-STAT3-S727, P-I?B-a, P-p65, p65, P-c-Jun, P-Akt, P-ERK1/2, P-p38, and P-SAPK/JNK
Farhang Ghahremani et al., Cell Death Differ 2013 (Anoxia...) : Unexpectedly, and for the first time, we demonstrate that p53 rapidly induces VEGF transcription upon hypoxia exposure by binding, in an HIF-1a dependent manner, to a highly conserved and functional p53 binding site within the VEGF promoter
Lee et al., J Biol Chem 2013 (Colonic Neoplasms) : These results identify KLF5 as a transactivator of HIF-1a and show that LPA regulates HIF-1a by dynamically modulating its interaction with KLF5 and p53