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IFI44 — POLDIP2

Text-mined interactions from Literome

Morigiwa et al., Nihon Yakurigaku Zasshi 2000 : Release of cytokines may be induced via Ca2+ influx and activation of NFAT, NF-kappa B or p44/42 and p38 MAP kinases, switching off the mitotic signal transduction pathway and triggering the apoptotic cascade at the same time
Tokuda et al., J Endocrinol 2001 : These results strongly suggest that p44/p42 MAP kinase activation is not involved in the PGE1 stimulated VEGF synthesis in osteoblasts but that p38 MAP kinase activation is involved
Takata et al., Br J Pharmacol 2001 : 3. Pravastatin inhibited the p44/42 MAP kinase activity induced by thrombin, but did not inhibit the p38 MAP kinase activity
Vela et al., Mol Cell Neurosci 2002 : Regarding intracellular signaling, IL-1beta activated the p38 mitogen activated protein kinase ( MAPK ) but not the p42/p44 MAPK and, when combined with growth factors, intensified p38 activation but inhibited the growth-factor induced p42/p44 activation
Sheng et al., J Neuroimmune Pharmacol 2009 (MAP Kinase Signaling System) : IL-1beta triggered the activation of p38 and ERK1/2 ( p44/42) MAP kinase (MAPK) signaling pathways, but WIN55,212-2 mainly inhibited p38 MAPK phosphorylation