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NGF — RNF19A

Text-mined interactions from Literome

Zhang et al., Neurobiol Aging 1999 (Alzheimer Disease...) : Oxidative stress differentially modulates phosphorylation of ERK, p38 and CREB induced by NGF or EGF in PC12 cells
Minneman et al., J Neurochem 2000 : p38 mitogen activated protein kinase inhibition reduced most norepinephrine responses, but not NGF responses
Burry et al., J Neurosci Res 2001 (MAP Kinase Signaling System) : However, NGF stimulated phosphorylation of ERK, p38 , and Akt in PC12-N09rasWT cells is similar in duration to that in PC12-N09 cells, indicating that the p21(ras) signaling through ERK, p38, and Akt was not involved in the restoration of normal neurite elongation in PC12-N09 cells
Arien-Zakay et al., J Mol Neurosci 2007 (Neuroblastoma) : Combined CM and NGF treatment induced constitutive activation of the mitogen activated protein kinases ERK2 ( 36-fold vs control ), p38alpha ( nine-fold vs control ) and p38beta ( 23-fold vs control ), most likely related to survival and/or differentiation
Yung et al., Cell Signal 2008 (MAP Kinase Signaling System) : NGF dependent p38 MAPK phosphorylation became insensitive to PTX treatment upon transient expressions of Galpha ( z ) or the PTX-resistant mutants of Galpha ( i2 ) and Galpha ( oA )