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POLDIP2 — RNF19A

Text-mined interactions from Literome

Nagano et al., Int Immunol 2002 : Taken together, these results suggest that both ERK and p38 ( MAPK ) pathways are involved in LPS induced COX-2 expression and PGE ( 2 ) production in neutrophils, and IL-10 and IL-4 inhibit neutrophil prostanoid synthesis by down regulating the activation of p38 ( MAPK )
Kim et al., Am J Physiol Lung Cell Mol Physiol 2005 : Additionally, we observed that UfCP exposure induces the phosphorylation and activation of p38 mitogen activated protein kinase ( MAPK ) in a biphasic manner and that the inhibition of p38 MAPK activity can block IL-8 mRNA expression induced by UfCP in NHBE cells
Cheng et al., Biotechnol Appl Biochem 2006 : The highly specific p38- or JNK-MAPK inhibitors strongly reduced respectively the p38- or JNK-like MAPK phosphorylation, which also demonstrated that there were no cross-reactions between the p38-like MAPK and JNK-like MAPK in T. cuspidata cells
Shastry et al., Kidney Int 2007 : We conclude that in MC, Hcy ( i ) activates p38-MAPK and increases p38MAPK nuclear translocation via an oxidative stress dependent mechanism and ( ii ) induces DNA damage and apoptosis that is dependent on oxidative stress and p38-MAPK activation
Goleva et al., Am J Respir Cell Mol Biol 2009 : The presence of the p38 MAPK inhibitor, SB203580, or IFN-gamma inhibited p38 MAPK phosphorylation and enhanced GCRalpha nuclear translocation in response to DEX
Chu et al., Can J Neurol Sci 2012 (Disease Models, Animal...) : Here we demonstrate that ( 1 ) both HS014 and SB203580 reduced CCI reduced hyperalgesia ( 2 ) p-p38MAPK was increased after CCI with a time course parallel to that of the MC4R change, ( 3 ) The p38 activation was prevented by blocking MC4R with an antagonist HS014, but MC4R-IR was not prevented by SB203580