◀ Back to VEGFA

ESR1 — VEGFA

Text-mined interactions from Literome

Qin et al., Clin Cancer Res 2001 (Breast Neoplasms...) : Association of ARNT splice variants with estrogen receptor negative breast cancer, poor induction of vascular endothelial growth factor under hypoxia, and poor prognosis
Kawai et al., Oncogene 2002 (Breast Neoplasms...) : Direct interaction between BRCA1 and the estrogen receptor regulates vascular endothelial growth factor ( VEGF ) transcription and secretion in breast cancer cells ... Furthermore, ER-alpha induced activation of VEGF gene transcription, using human VEGF promoter-luciferase reporter constructs
Banerjee et al., Neoplasia (New York, N.Y.) 2003 (Breast Neoplasms...) : Here we describe a regulatory role of vascular endothelial growth factor-A ( VEGF-A ) in the biphasic effects on estrogen receptor ( ER ) + GH3 rat pituitary tumor cells and MCF-7 human breast tumor cells depending on the dosage of 2-ME2 used
Hamada et al., Circulation 2006 (Myocardial Infarction) : Moreover, ER alpha contributes to upregulation of vascular endothelial growth factor , revealing possible mechanisms of an effect of E2 on EPC biology
Bake et al., Endocrinology 2008 : Estrogen receptor-alpha overexpression suppresses 17beta-estradiol mediated vascular endothelial growth factor expression and activation of survival kinases
Banerjee et al., Clin Cancer Res 2008 (Breast Neoplasms) : Estrogen bound estrogen receptor enhances VEGF expression, providing a common link between these signaling pathways that may be targeted by endocrine therapy
Wang et al., J Thorac Cardiovasc Surg 2009 (MAP Kinase Signaling System) : Signal transducer and activator of transcription 3-stimulated hypoxia inducible factor-1alpha mediates estrogen receptor-alpha induced mesenchymal stem cell vascular endothelial growth factor production ... Signal transducer and activator of transcription 3 knockout abolished estrogen receptor-alpha induced hypoxia inducible factor-1alpha and subsequent vascular endothelial growth factor production
Hyder et al., Cancer Res 1996 : E2-dependent VEGF induction is inhibited by actinomycin D but not puromycin, suggesting that the effect is due at least in part to direct estrogen receptor regulation of VEGF transcription