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CREB1 — NOS1

Text-mined interactions from Literome

Mishra et al., Neuroscience 2002 (Hypoxia-Ischemia, Brain) : The present study tests the hypothesis that inhibition of NOS by N-nitro-L-arginine ( NNLA ) will prevent the hypoxia induced increased phosphorylation of CREB protein in neuronal nuclei of newborn piglets ... The data show that NOS inhibition attenuates the hypoxia induced increase in CREB protein phosphorylation in the cerebral cortex of newborn piglets
Park et al., Neuroreport 2004 : The inhibition of nitric oxide synthase enhances PSA-NCAM expression and CREB phosphorylation in the rat hippocampus
Mantelas et al., Brain Res 2007 : These results indicate that the following mechanism could be operating in a gonadal hormone dependent and brain area-specific manner during neonatal rat brain development : Depolarization following GABA-A receptor activation leads to opening of L-type voltage gated calcium channels, resulting in an increased Ca ( 2+ ) influx, which in turn leads to phosphorylation, and thus activation of the transcription factor CREB ; the phosphorylated CREB can then induce nNOS
Gutsaeva et al., J Neurosci 2008 (Anoxia) : These findings demonstrate that hypoxic preconditioning elicits subcortical mitochondrial biogenesis by a novel mechanism that requires nNOS regulation of PGC-1alpha and CREB
Gallo et al., J Neurosci 2011 : NOS inhibition did not affect phosphorylation of cAMP response element binding protein ( CREB ), a well established ERK nuclear target, but it attenuated the nuclear accumulation of the CREB coactivator TORC1 and suppressed the activation of Elk-1, another transcription factor target of ERK
Ding et al., J Neurosci 1997 : Inhibition of NOS during Glu stimulation diminished the anti-P-CREB-lir of this 43 kDa band