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APOB — LOX

Text-mined interactions from Literome

Rodríguez et al., Arterioscler Thromb Vasc Biol 2002 (Arteriosclerosis...) : Inhibition of LOX activity by either LDL or beta-aminopropionitrile, an inhibitor of LOX , increased endothelial permeability ( 192+/-0.19- and 3.37+/-0.74-fold, respectively )
Mukai et al., Atherosclerosis 2004 : Lectin-like Ox-LDL receptor-1 ( LOX-1 ) is highly inducible by proinflammatory cytokines, as well as angiotensin II and Ox-LDL in vitro
Ding et al., Biochem Biophys Res Commun 2012 (Atherosclerosis) : Primary human umbilical vein endothelial cells ( HUVECs ) were used to study the expression of lectin-like ox-LDL receptor-1 ( LOX-1 ) , HSPG, autophagy and apoptosis in response to ox-LDL and heparinase III (Hep III) ... Importantly, Hep III pretreatment of cells significantly enhanced ox-LDL induced HSPG degradation, LOX-1 expression, ox-LDL uptake and ROS generation as well as autophagy and apoptosis