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AHR — PDCD11

Text-mined interactions from Literome

Hardin et al., Toxicol Appl Pharmacol 1992 : The results support the hypothesis that B [ a ] P suppression of B cell lymphopoiesis is mediated at least in part by the induction of programmed cell death and that the Ah receptor and/or P450 isoenzymes are involved in this process
Vogel et al., Am J Pathol 2007 (Lymphoma) : Here, we show that activation of the aryl hydrocarbon receptor (AhR) by 2,3,7,8-tetrachlorodibenzo-p-dioxin ( TCDD ) resulted in loss of the programmed cell death ( apoptosis ) response in three different lymphoma cell lines, which plays a key role in the development of cancer, especially lymphoma and leukemia
Chopra et al., Crit Rev Toxicol 2011 : Dioxin toxicity, aryl hydrocarbon receptor signaling, and apoptosis-persistent pollutants affect programmed cell death