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CASP9 — CD79A

Text-mined interactions from Literome

Nitta et al., Exp Cell Res 2001 : Furthermore, addition of spermine could repress the BCR mediated apoptosis by attenuating the mitochondrial membrane potential ( Deltapsim ) loss and activation of caspase-7 induced by BCR signaling
Kim et al., Br J Haematol 2004 (Leukemia, Myelogenous, Chronic, BCR-ABL Positive) : Imatinib/apicidin co-treatment for 48 h produced a prominent decrease in Bcr-Abl protein levels in a caspase dependent manner ... In summary, these data indicate that apicidin potentiates the imatinib induced apoptosis of Bcr-Abl positive leukaemia cells through the enhanced activation of the mitochondria dependent caspase cascades, accompanied by caspase dependent downregulation of Bcr-Abl and XIAP
Deming et al., Mol Cell Biol 2004 : We report here that Bcr-Abl also inhibits caspase activation after the release of cytochrome c. Bcr-Abl inhibited caspase activation by cytochrome c added to cell-free lysates and prevented apoptosis when cytochrome c was microinjected into intact cells
Ichinohe et al., J Exp Med 2009 (Orthomyxoviridae Infections) : Although NLRP3 was required for inflammasome activation in certain cell types, CD4 and CD8 T cell responses, as well as mucosal IgA secretion and systemic IgG responses, required ASC and caspase-1 but not NLRP3
Fei et al., Zhonghua Er Ke Za Zhi 2013 (Purpura, Schoenlein-Henoch) : Moreover IgA1 isolated from HSP patients could significantly increase the caspase-3 and Fas expression ( P < 0.01 )
Lens et al., J Immunol 1998 : Indeed, ligation of both Fas and BCR resulted in cleavage of the IL-1beta converting enzyme/Ced-3-like protease caspase 3 and its substrates Ac-Asp-Glu-Val-Asp-aldehyde and poly ( ADP-ribose ) polymerase