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APOB — CSF1

Text-mined interactions from Literome

Fuhrman et al., Atherosclerosis 2008 : We thus conclude that Ox-LDL induces monocyte differentiation to macrophages in vivo and this phenomenon involves activation of the M-CSF-receptor
Ishibashi et al., J Biol Chem 1990 : Binding of LDL was also enhanced by M-CSF but less significantly than that of acetyl-LDL
Rajavashisth et al., Arterioscler Thromb Vasc Biol 1995 : Nuclear runon assays showed that M-CSF gene transcription was activated by MM-LDL
Watanabe et al., Ann N Y Acad Sci 1995 (Arteriosclerosis) : M-CSF ( 100 ng/ml ) enhanced acidic and neutral CE hydrolase, and ACAT activities by 3.2-fold, 4-fold, and 2.3-fold, respectively, in the presence of acetyl LDL , and M-CSF increased ratios of both acidic and neutral CE hydrolase activities to ACAT activity
Suzu et al., J Clin Invest 1994 (Arteriosclerosis) : The addition of exogenous chondroitin sulfate or apolipoprotein B inhibited the binding of PG-M-CSF to LDL in a dose dependent manner, indicating that the interaction between PG-M-CSF and LDL was mediated by the binding of the chondroitin sulfate chain of PG-M-CSF to LDL apolipoprotein B
Pai et al., Kidney Int 1995 : Since atherogenic lipoproteins ( such as low density lipoprotein, LDL ) have been implicated in monocyte migration and proliferation into the glomerular mesangium, we examined the effect of LDL on mesangial cell expression of macrophage colony stimulating factor ( M-CSF ), a cytoregulatory peptide associated with monocyte chemoattraction, differentiation and proliferation