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CAT — GSR

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Afonso et al., Free radical research 1999 : Increased levels of hydrogen peroxide coming from the reaction catalyzed by SOD and lipid peroxides as a consequence of membrane peroxidation, induced the activity of catalase and glutathione peroxidase ( GPx ), while decreased GSH levels induced glutathione reductase (GRed) activity
Kim et al., J Pharm Pharmacol 2000 : Both MA-11 and PT-11, preserved the levels of catalase and inhibited decreases in glutathione reductase in glutamate injured cells
Parida et al., J Plant Physiol 2004 : In the leaves of B. parviflora, salt treatment preferentially enhanced the content of H2O2 as well as the activity of ascorbate peroxidase ( APX ), guaiacol peroxidase ( GPX ), glutathione reductase (GR) , and superoxide dismutase ( SOD ), whereas it induced the decrease of total ascorbate and glutathione ( GSH+GSSG ) content as well as catalase (CAT) activity
Fraga et al., International journal of biological sciences 2013 : However, superoxide dismutase was not affected by poly P15, catalase activity was stimulated only at high concentrations and glutathione reductase was the only enzyme that was stimulated in the same way by both poly Ps. Altogether, our results indicate that inorganic polyphosphate and mitochondrial membrane exopolyphosphatase regulation can be correlated with the generation of reactive oxygen species in the mitochondria of R. microplus embryos
Chandra et al., Int J Cardiol 1994 (Myocardial Ischemia) : Subset analysis revealed that in unstable angina and acute myocardial infarction, superoxide anion, malonyldialdehyde and glutathione reductase were elevated while superoxide dismutase and catalase levels were reduced
Tatsumi et al., Basic Res Cardiol 1993 : Combined treatment of myocytes with 3-amino-1,2,4-triazole, 1,3-bis ( 2-chloroethyl ) -1-nitrosourea and diethyl maleate ( to inactivate catalase , to inhibit glutathione reductase activity, and to deplete glutathione, respectively ) enhanced the sensitivity of translocase to hydrogen peroxide, supporting the view that the cellular defense mechanism is a significant factor in determining the toxicity of hydrogen peroxide