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MET — PI3

Text-mined interactions from Literome

Nakanishi et al., Clin Exp Metastasis 1999 (Carcinoma, Hepatocellular) : HGF stimulated the motility of Hep3B, HepG2, and Huh-7 cells in a dose dependent manner in association with tyrosine phosphorylation of c-Met and activation of phosphatidylinositol 3-kinase (PI3-K)
Basar et al., Infect Immun 2005 : Binding of InlB leads to phosphorylation of Met and the adapter Gab1 and to activation of host phosphoinositide (PI) 3-kinase
Grotegut et al., Int J Oncol 2010 (Hepatoblastoma...) : Here, we demonstrate that HGF has no impact on overall cell viability and proliferation of HB cells, although signal transduction occurs downstream of HGF, such as c-Met phosphorylation, activation of phosphoinositide 3-kinase (PI3K)/AKT and mitogen activated protein kinase ( MAPK ) /ERK-1/2 signaling