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ATP5O — BCL2

Text-mined interactions from Literome

Kobrinsky et al., Oncogene 2001 : Evidence for a role of the sarcoplasmic/endoplasmic reticulum Ca ( 2+ ) -ATPase in thapsigargin and Bcl-2 induced changes in Xenopus laevis oocyte maturation
Ma et al., Sci China C Life Sci 2003 : These results indicated TMSG-1 up regulation in prostate cancer cell line could promote V-ATPase activity, increase pHi and cell apoptosis, and inhibit the expression of BCL2
Sasazawa et al., Cancer Sci 2009 (Lung Neoplasms...) : Our data suggest that V-ATPase inhibitors could not only suppress anti-apoptotic Bcl-2 nor Bcl-xL but could also facilitate the caspase independent apoptotic pathway
Matsumoto et al., Dig Dis Sci 2011 : The VacA mediated reduction of STAT3 expression was independent of cellular vacuolization, since a vacuolar-type ATPase inhibitor, bafilomycin A1, did not inhibit VacA induced reduction of STAT3, Bcl-2, and Bcl-XL expression ... The VacA mediated reduction of STAT3 expression was independent of cellular vacuolization, since a vacuolar-type ATPase inhibitor, bafilomycin A1, did not inhibit VacA induced reduction of STAT3, Bcl-2 , and Bcl-XL expression
Gilbert et al., J Cell Physiol 1997 (Lymphoma, B-Cell) : In summary, this data suggests that Bcl-2 affects Na+/K ( + ) -ATPase pump activity, which is associated with the resting membrane potential and the level of susceptibility to radiation induced PCD