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CISH — IL10

Text-mined interactions from Literome

Ito et al., Blood 1999 : This may occur through the ability of IL-10 to induce expression of the gene, suppressor of cytokine signaling 3 ( SOCS3 )
Cassatella et al., Blood 1999 : Interleukin-10 (IL-10) selectively enhances CIS3/SOCS3 mRNA expression in human neutrophils : evidence for an IL-10 induced pathway that is independent of STAT protein activation
Ding et al., J Immunol 2003 : IL-10 induced SOCS is associated with the inhibition of IFN-gamma signaling in various cell types, and this inhibition is independent of C-terminal serine residues of the IL-10R, previously shown to be required for other anti-inflammatory responses
Cassatella et al., Eur J Immunol 2005 : Activation of signal transducer and activator of transcription 3 ( STAT3 ) phosphorylation and induction of suppressor of cytokine signaling (SOCS)-3 protein by IL-10 are in fact negligible in circulating or freshly isolated ( `` time 0 '' ) neutrophils, but become readily measurable in neutrophils cultured for 4 h in the presence or absence of LPS
Frobøse et al., Mol Endocrinol 2006 : Suppressor of cytokine Signaling-3 inhibits interleukin-1 signaling by targeting the TRAF-6/TAK1 complex
Dennis et al., Infect Immun 2006 (Lyme Disease) : Because it is known that SOCS are induced by IL-10 and that B. burgdorferi and its lipoproteins most likely interact via TLR2 or the heterodimers TLR2/1 and/or TLR2/6, we hypothesized that SOCS are induced by IL-10 and B. burgdorferi and its lipoproteins in macrophages and that SOCS may mediate the inhibition by IL-10 of concomitantly elicited cytokines
Prêle et al., J Immunol 2008 : In addition, SOCS1 regulated the production of IL-6, but not IL-10 , by monocytes
Renner et al., Nutrients 2013 : Quantitative real time polymerase chain reaction analyses were performed to evaluate the expression of interleukin (IL)-4, IL-10 , interferon ( IFN ) -?, tumor necrosis factor (TNF)-a and peroxisome proliferator activated receptor ( PPAR ) -? in response to cis-9,trans-11 and LA
Holán et al., J Immunol 1998 : The principal cell population displaying the cis-UCA induced elevated production of IL-10 was CD4+ T cells, which were shown to be a direct target of cis-UCA action