We have a suspicion that you are an automated web bot software, not a real user. To keep our site fast for other users, we have slowed down this page. The slowdown will gradually disappear. If you think this is a mistake, please contact us at genome-www@soe.ucsc.edu. Also note that all data for hgGeneGraph can be obtained through our public MySQL server and all our software source code is available and can be installed locally onto your own computer. If you are unsure how to use these resources, do not hesitate to contact us.
UCSC Genome Browser Gene Interaction Graph
Gene interactions and pathways from curated databases and text-mining

◀ Back to EPO

EPO — GSK3B

Text-mined interactions from Literome

Wu et al., Apoptosis 2007 : The effects of EPO on GSK-3beta and caspase-3 activities were also blocked by LY294002
Nishihara et al., J Mol Cell Cardiol 2007 (Disease Models, Animal...) : To get an insight into the mechanism by which phospho-GSK-3beta protects the myocardium from ischemia/reperfusion injury, we examined the effects of IPC and EPO on interactions between GSK-3beta and subunits of the mitochondrial permeability transition pore ( mPTP ) in this study ... IPC+EPO significantly increased the levels of GSK-3beta and phospho-GSK-3beta that were co-immunoprecipitated with ANT to 145+/-8 % and 143+/-16 %, respectively, of baseline but did not induce phospho-GSK-3beta-VDAC binding ... A PKC inhibitor and a PI3 kinase inhibitor suppressed the IPC+EPO induced increase in the level of phospho-GSK-3beta-ANT complex
Ohori et al., Am J Physiol Heart Circ Physiol 2008 : However, EPO increased the level of Ser9-phospho-GSK-3beta colocalized with mitochondria by 50 % in a phosphatidylinositol 3-kinase dependent manner
Miki et al., Diabetes 2009 (Body Weight...) : However, neither phosphorylation of Akt and GSK-3beta nor infarct size limitation was induced by EPO in OLETF rats